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Hypopituitarism
Wikipedia
The pituitary gland was very small and there were few remnants of both the anterior and the posterior pituitary. [1] [5] The eponym Simmonds' syndrome is used infrequently for acquired hypopituitarism, especially when cachexia (general ill health and malnutrition) predominates. [27] [28] Most of the classic causes of hypopituitarism were described in the 20th century; the early 21st century saw the recognition of how common hypopituitarism could be in previous head injury victims. [1] Until the 1950s, the diagnosis of pituitary disease remained based on clinical features and visual field examination, sometimes aided by pneumoencephalography and X-ray tomography . ... "Hypothalamic hormones a.k.a. hypothalamic releasing factors" . J. Endocrinol . 184 (1): 11–28. doi : 10.1677/joe.1.05883 . PMID 15642779 . ^ a b c Arafah BM, Nasrallah MP (December 2001).LHX4, FOXA2, PRL, CYP17A1, CYP19A1, HESX1, PROP1, LHX3, SMPD3, CSHL1, KISS1R, NR5A1, KISS1, GHRH, TACR3, GLI2, GNRH1, GNRHR, GPC1, SOX3, ALX3, GH1, MAGEL2, ZSWIM6, APOA5, SOX2, POU1F1, POMC, PROKR2, IGF1, SLC20A1, CTNNB1, SST, HTC2, OTX2, AR, AVP, PROK2, SRY, EIF2S3, ECE2, ARNT2, CDON, CDK4, PACC1, CHD7, SHOC2, BMP2, RSS, SBE2, FLNA, L1CAM, GAS1, ACSM3, NFKB2, NONO, PAX6, APOE, ANOS1, IGSF1, SIM1, GHRHR, SIX3, SMS, SOD1, GPT, SSRP1, LAMB2, EEF1AKMT4-ECE2
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Female Infertility
Wikipedia
Some authors suggest that physicians should investigate the presence of undiagnosed coeliac disease in women with unexplained infertility, recurrent miscarriage or IUGR. [25] [26] Significant liver or kidney disease Thrombophilia [27] [28] Cannabis smoking , such as of marijuana causes disturbances in the endocannabinoid system , potentially causing infertility [29] Radiation, such as in radiation therapy . ... "Fertility after ectopic pregnancy: The DEMETER randomized trial" . Human Reproduction . 28 (5): 1247–1253. doi : 10.1093/humrep/det037 .CYP19A1, AR, LIF, ADIPOR2, ADIPOR1, CACNA2D2, NR1I2, TP53, NR2F2, SMO, PRL, MAPK3, MAPK1, ARNTL, MTHFR, PAPPA, LHB, CEBPA, LEP, NR5A2, FSHB, ESR2, ESR1, DHFR, CYP17A1, LEPR, CCN2, CEBPB, TUBB8, TLE6, FOXL2, ZP2, PANX1, STAG3, HUWE1, PGR, CASTOR3
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Dysthymia
Wikipedia
These tactics may be helpful in warding off or delaying dysthymic symptoms. [28] Treatments [ edit ] Main article: Management of depression Persistent depressive disorder can be treated with psychotherapy and pharmacotherapy .
- Leptin Deficiency Or Dysfunction Omim
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Visual Impairment
Wikipedia
It has been estimated that over half of completely blind people have non-24-hour sleep–wake disorder , a condition in which a person's circadian rhythm , normally slightly longer than 24 hours, is not entrained (synchronized) to the light–dark cycle. [27] [28] Cause [ edit ] The most common causes of visual impairment globally in 2010 were: Refractive error (42%) Cataract (33%) Glaucoma (2%) Age-related macular degeneration (1%) Corneal opacification (1%) Diabetic retinopathy (1%) Childhood blindness Trachoma (1%) Undetermined (18%) [8] The most common causes of blindness worldwide in 2010 were: Cataracts (51%) Glaucoma (8%) Age-related macular degeneration (5%) Corneal opacification (4%) Childhood blindness (4%) Refractive errors (3%) Trachoma (3%) Diabetic retinopathy (1%) Undetermined (21%) [8] About 90% of people who are visually impaired live in the developing world . [5] Age-related macular degeneration, glaucoma, and diabetic retinopathy are the leading causes of blindness in the developed world. [29] Among working-age adults who are newly blind in England and Wales the most common causes in 2010 were: [30] Hereditary retinal disorders (20.2%) Diabetic retinopathy (14.4%) Optic atrophy (14.1%) Glaucoma (5.9%) Congenital abnormalities (5.1%) Disorders of the visual cortex (4.1%) Cerebrovascular disease (3.2%) Degeneration of the macula and posterior pole (3.0%) Myopia (2.8%) Corneal disorders (2.6%) Malignant neoplasms of the brain and nervous system (1.5%) Retinal detachment (1.4%) Cataracts [ edit ] Cataracts are the congenital and pediatric pathology that describes the greying or opacity of the crystalline lens, which is most commonly caused by intrauterine infections, metabolic disorders, and genetically transmitted syndromes. [31] Cataracts are the leading cause of child and adult blindness that doubles in prevalence with every ten years after the age of 40. [32] Consequently, today cataracts are more common among adults than in children. [31] That is, people face higher chances of developing cataracts as they age.TTPA, TSEN2, ATXN7, TSEN54, TSEN34, CLN6, HK1, WHRN, FOXP1, TBL2, CNNM4, PTPN22, SETBP1, NSMF, MMACHC, NDUFAF3, ABCA1, FSCN2, TUBGCP4, AIPL1, AMACR, LEMD3, ZFYVE26, SF3B1, CRB1, ADNP, SMCHD1, FLRT3, COQ2, PLXND1, GLRX5, NCAPG2, BCOR, AHI1, TRIM44, IL17RD, CNGB3, GTPBP2, NDUFB11, PIGT, PCDH12, TMEM216, SLC45A2, OSTM1, SAR1B, MECR, POLR1D, SOST, POMT2, GMPPA, GMPPB, SNX10, ANKRD11, UBE2T, FLVCR1, RPGRIP1L, SNRNP200, TTLL5, RAB11B, KIAA0586, IFT140, GDF3, GTF2IRD1, POLR1C, PEX16, HS6ST1, RAB28, ZNHIT3, AIMP1, LARGE1, ARNT2, AIFM1, USP8, UNC119, BAZ1B, AP3D1, BCL10, ST3GAL5, SYNJ1, PROM1, FGF17, PLEKHM1, SCO2, RIMS1, CIB2, ATF6, ARSG, EXOSC8, TREX1, PRDM5, CLP1, MALT1, PLK4, POMT1, SLC19A2, MAD2L2, USH1C, YAP1, TUBB4A, TUBB3, SEMA3A, TFG, IKZF1, LAMC3, TCIRG1, COQ7, TSFM, MKS1, RFWD3, FANCL, C12orf65, SFXN4, TWIST2, NUS1, C1QTNF5, SLC52A3, TP53RK, CEP41, CACNA2D4, CDHR1, RFT1, TMEM67, EARS2, TUBGCP6, ALG2, USP45, RAX2, KISS1R, ZNF469, SLX4, HPS3, PHF6, ANTXR1, ISCA2, USH1G, ADGRV1, MFSD8, SDHAF1, GDF6, FEZF1, PCARE, BLOC1S3, RGS9BP, NHLRC1, SUMF1, CCDC141, POC1B, IBA57, MICOS13, ARX, LCA5, VPS13B, C8orf37, CEP120, CKAP2L, B3GALNT2, B3GLCT, PROKR2, DRAM2, CEP78, BRIP1, SUCLA2, WDR45B, CC2D2A, ARID1B, TBC1D24, MCOLN1, SALL4, CYSLTR2, RPGRIP1, CABP4, MFF, INPP5E, ANKH, PROK2, SMG9, SPATA7, MBD5, WDR11, PEX26, OSGEP, CHD7, SLC39A4, POMGNT1, FANCI, FANCM, TRAPPC11, SLC4A11, SRD5A3, PITPNM3, SPRY4, ADAMTS10, COL18A1, SPG11, OPA3, CEP290, PDZD7, CSPP1, PALB2, CARS2, PIEZO2, SLC52A2, FKRP, PCDH15, NMNAT1, SEMA4A, XYLT2, NOD2, CDH23, PRDM16, ELMO2, HESX1, RGS9, PEX11B, CYTB, HBB, HARS1, HADHA, HSD17B10, GUCY2D, GUCA1B, GUCA1A, GTF2I, GSN, GRN, GNAT2, GNAQ, GNA11, GJA1, OPN1MW, GATA2, GABRD, FOXE3, FOXC1, FH, FGFR2, FGFR3, FGFR1, HCCS, HLA-A, HLA-B, LCAT, ATP6, MITF, MC1R, MAB21L1, LTBP2, LSS, LRPAP1, LRP5, LMNB1, LIMK1, LAMP2, HLA-DPA1, KIF11, ANOS1, ITPR1, IMPDH1, IGH, IGBP1, HSPG2, HSD17B4, HLA-DQB1, HLA-DPB1, FGF8, FDXR, FKTN, BRCA2, COL4A1, COL2A1, CNGA3, CLCN7, LYST, CAMK2B, CACNA1F, CA2, CFAP410, BTD, BRCA1, COL17A1, BDNF, KIF1A, RERE, ASPA, PHOX2A, ABCC6, FAS, BIRC3, AIRE, ABCD1, COL7A1, COX7B, EFEMP1, ERCC4, FBN1, FANCG, FANCF, FANCB, FANCE, FANCD2, FANCC, FANCA, ERCC6, ERCC5, ERCC2, CRX, ERCC1, ELN, DUSP6, TIMM8A, DDX3X, DCC, CYP1B1, CTSD, CTLA4, VCAN, COX3, ND1, ABCA4, ND2, TTR, TP53, TK2, TGFBI, TEK, TCOF1, TAT, TACR3, SYT1, SURF1, SRY, SOX10, SOX3, SOX2, SMARCB1, SMARCA4, SKI, SDHD, SDHA, SAG, RPE65, RPGR, RMRP, TYR, USH2A, CLRN1, PLA2G6, ADAM9, GPAA1, TNFSF11, CASK, PDXK, AP3B1, IKBKG, KCNAB2, CNTNAP1, PEX3, BAP1, VHL, TRRAP, ARID1A, NAA10, AAAS, EPM2A, ALMS1, XRCC2, WT1, CLIP2, BEST1, RFC2, REV3L, PRPH2, NDUFB8, OPA1, OCRL, OCA2, GPR143, NTRK2, NRL, NOTCH3, NHS, NF2, NDUFS2, MYOC, PRDX1, MYO7A, TRNS2, TRNL1, TRNK, TRNE, MTTP, ND6, ND5, ND4L, ND4, OTX2, PAX2, OPN1LW, POLA1, RAD51C, RAD51, PEX5, PEX2, PEX19, PRTN3, PRPS1, PRNP, PPP2CA, POLG, PLOD1, PAX6, PEX14, PEX13, PEX12, PEX10, PEX6, PEX1, PEPD, ENPP1, PDE6C, PCYT1A, RNU4ATAC
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Obstetric Fistula
Wikipedia
A patient's incontinence and pain also render her unable to perform household chores and childrearing as a wife and as a mother, thus devaluing her. [24] Other misconceptions about obstetric fistulae are that they are caused by venereal diseases or are divine punishment for sexual misconduct. [25] As a result, many girls are divorced or abandoned by their husbands and partners, disowned by family, ridiculed by friends, and even isolated by health workers. [17] Divorce rates for women who suffer from an obstetric fistula range from 50% [26] [27] [28] [29] to as high as 89%. [25] Now marginalized members of society, girls are forced to live on the edges of their villages and towns, often to live in isolation in a hut where they will likely die from starvation or an infection in the birth canal.
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Histrionic Personality Disorder
Wikipedia
I look forward to talking to you. [26] Functional ideographic assessment template [ edit ] Another example of treatment besides coding is functional ideographic assessment template. [26] The functional ideographic assessment template, also known as FIAT, was used as a way to generalize the clinical processes of functional analytic psychotherapy. [26] The template was made by a combined effort of therapists and can be used to represent the behaviors that are a focus for this treatment. [26] Using the FIAT therapists can create a common language to get stable and accurate communication results through functional analytic psychotherapy at the ease of the client; as well as the therapist. [26] Epidemiology [ edit ] The survey data from the National epidemiological survey from 2001–2002 suggests a prevalence of HPD of 1.84 percent. [27] [ medical citation needed ] Major character traits may be inherited, while other traits may be due to a combination of genetics and environment, including childhood experiences. [8] This personality is seen more often in women than in men. [28] Approximately 65% of HPD diagnoses are women while 35% are men.
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Traumatic Brain Injury
Wikipedia
A current model developed by the Department of Defense and Department of Veterans Affairs uses all three criteria of GCS after resuscitation , duration of post-traumatic amnesia (PTA), and loss of consciousness (LOC). [20] It also has been proposed to use changes that are visible on neuroimaging , such as swelling , focal lesions, or diffuse injury as method of classification. [9] Grading scales also exist to classify the severity of mild TBI, commonly called concussion ; these use duration of LOC, PTA, and other concussion symptoms. [23] Pathological features Main article: Focal and diffuse brain injury CT scan Spread of the subdural hematoma (single arrows), midline shift (double arrows) Systems also exist to classify TBI by its pathological features. [19] Lesions can be extra-axial, (occurring within the skull but outside of the brain) or intra-axial (occurring within the brain tissue). [24] Damage from TBI can be focal or diffuse , confined to specific areas or distributed in a more general manner, respectively. [25] However, it is common for both types of injury to exist in a given case. [25] Diffuse injury manifests with little apparent damage in neuroimaging studies, but lesions can be seen with microscopy techniques post-mortem , [25] [26] and in the early 2000s, researchers discovered that diffusion tensor imaging (DTI), a way of processing MRI images that shows white matter tracts, was an effective tool for displaying the extent of diffuse axonal injury . [27] [28] Types of injuries considered diffuse include edema (swelling), concussion and diffuse axonal injury, which is widespread damage to axons including white matter tracts and projections to the cortex . [29] [30] Focal injuries often produce symptoms related to the functions of the damaged area . [17] Research shows that the most common areas to have focal lesions in non-penetrating traumatic brain injury are the orbitofrontal cortex (the lower surface of the frontal lobes) and the anterior temporal lobes , areas that are involved in social behavior, emotion regulation, olfaction, and decision-making, hence the common social/emotional and judgment deficits following moderate-severe TBI. [31] [32] [33] [34] Symptoms such as hemiparesis or aphasia can also occur when less commonly affected areas such as motor or language areas are, respectively, damaged. [35] [36] One type of focal injury, cerebral laceration , occurs when the tissue is cut or torn. [37] Such tearing is common in orbitofrontal cortex in particular, because of bony protrusions on the interior skull ridge above the eyes. [31] In a similar injury, cerebral contusion (bruising of brain tissue), blood is mixed among tissue. [22] In contrast, intracranial hemorrhage involves bleeding that is not mixed with tissue. [37] Hematomas, also focal lesions, are collections of blood in or around the brain that can result from hemorrhage. [10] Intracerebral hemorrhage , with bleeding in the brain tissue itself, is an intra-axial lesion.
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Mercury Poisoning
Wikipedia
The toxic damage appears to be determined by the peak value of mercury, not the length of the exposure. [27] Methylmercury exposure during rodent gestation, a developmental period that approximately models human neural development during the first two trimesters of gestation, [28] [29] has long-lasting behavioral consequences that appear in adulthood and, in some cases, may not appear until aging. ... S2CID 31630319 . ^ a b c Chris Stewart (22 January 2017). "#116-Tang 28: I, Xuānzong" . The History of China (Podcast). ... Archived from the original on 2011-08-28. ^ "An article about gilding" . Archived from the original on 2013-10-23. ^ "Bichloride of Mercury Killed Olive Thomas" . ... Archived from the original on 2012-11-08 . Retrieved 2007-01-28 . ^ Swearengin M (2008-04-01). "Man dies from mercury poisoning after trying to extract gold".
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Acute Pancreatitis
Wikipedia
In patients with severe volume depletion that manifests as hypotension and tachycardia, more rapid repletion with 20 mL/kg of intravenous fluid given over 30 minutes followed by 3 mL/kg/hour for 8 to 12 hours. [27] [28] Fluid requirements should be reassessed at frequent intervals in the first six hours of admission and for the next 24 to 48 hours.LEP, SERPINA1, REG3A, BIRC5, MTNR1B, CX3CL1, KIT, APOE, LMNA, CXCL8, IL10, PRSS1, BSCL2, CFTR, NSMCE2, BTBD8, MPO, CRP, AGPAT2, IL6, NLRP3, HMGCL, HMGB1, TNF, SPINK1, IL1B, TLR4, DPP4, ALB, CCL2, MAPK14, STAT3, PRSS2, GABPA, NFE2L2, AHSA1, PIK3CG, CRK, AKT1, MAPK1, AIMP2, CD14, LPL, GRAP2, HMOX1, RNF19A, ACE, POLDIP2, PIK3CD, PIK3CA, DSPP, TRAF3, PIK3CB, CALCA, JAK2, SH2D1A, CCK, APCS, IL1A, SST, MIR216A, GSTT1, GLP1R, GCG, GORASP1, MTAP, RIPK3, ELANE, WNK1, TLR9, CTRC, IL1RN, HSPA1B, SYT1, TGFB1, REN, PTX3, PTGS2, ACTB, ASPG, MAPK8, KMO, RELA, TAC1, MIR155, DECR1, IFNG, VEGFA, CLDN2, S100A9, TP53, SOCS3, SQSTM1, SETMAR, IFNA1, TLR3, HSPA2, RIPK1, HSPA4, ICAM1, TLR2, NOS2, KL, IL17A, NUP62, LILRB1, P2RX7, FGL2, PROCR, HSPA1A, PON1, MMP9, PPARGC1A, MEFV, MDK, CNPY2, PPID, PRKCD, LPA, LCN2, MPRIP, PTHLH, SIRT1, KHDRBS1, PYY, IFNA13, DCTN4, ANGPT1, CARD9, PTK2B, APOB, F2, MORC4, EHMT1, MIR551B, ASRGL1, TP53INP1, NQO1, APOA5, CASP9, CTNNB1, MLKL, CST3, CSF3, RTL1, GOLGA6A, GPIHBP1, ANGPT2, ERN1, CD34, GOT2, AGT, AGER, GTF2H1, RCBTB1, GPT, ALDH2, GSTM1, CBSL, VIP, MIR148A, OCLN, ADIPOQ, STING1, MIR141, AIM2, MIR127, GDF15, MIR122, IP6K1, NR1H4, ARFRP1, PPIF, VCAM1, MIR21, MTCO2P12, VDR, MAGI2, MALAT1, SRA1, LOC102724197, PLA2G7, MIR27A, BECN1, TFEB, MFT2, CD24, ZGLP1, CCR2, MANF, CXCR4, KMT2D, GALR3, SOCS1, TRPV1, RAMP1, RAB7A, DEFB4B, RIPK2, MIR542, GALR2, DEFB104B, MIR375, HSPB3, MIR339, CDKL2, PIM3, MIR34A, MIR29A, PIAS3, GADL1, RETN, ADM2, RAB21, TNIP2, TAB2, LMF1, TNMD, CELA3B, SMUG1, IL21, CXCL16, AKR1B10, SPHK2, FGF21, PARD3, BET1, SLC17A5, CCDC91, INTU, TREM1, SGSM3, UQCRFS1, GHRL, CD274, ERO1A, MBL3P, GAL, SARAF, ISYNA1, NINL, ADAMTS13, CPTP, VMP1, AKR1A1, RAB7B, OR10A4, TLR6, KLF2, RACK1, MARCHF2, GPRC6A, YAP1, MARVELD2, OLFM1, ATG7, GPBAR1, TICAM1, DEFB104A, NIBAN1, HPSE, B3GALT5-AS1, AZIN2, DNER, GGTLC1, IL33, UCN2, HAVCR2, RALBP1, SPINK5, COL25A1, UROD, ACR, UMOD, CTSG, ESR1, ERBB4, ERBB2, STX2, EPHB2, EPHB1, ENPEP, ELK3, EGR1, EGFR, EGF, DUSP1, RCAN1, DEFB4A, DEFB1, DDIT3, CYP24A1, ESR2, ETV5, F3, GH1, HGF, GSTP1, GSR, CXCL2, CXCL1, GP2, GLI2, GFER, F5, FUT2, FN1, FOXF1, FGFR1, FCGR3B, FCGR3A, FABP2, CYLD, CTSB, UCP2, CTH, SERPINC1, ARRB2, ARRB1, AQP5, FAS, APOC3, APOC2, AQP8, APOA1, XIAP, ANXA3, ANXA2, AHSG, AHCY, CRISP1, ADH5, ADH1C, RERE, AVP, BCL2, CD19, CRH, CLDN4, CPB2, CPB1, CNR2, LTB4R, CEL, CBS, BCL3, RUNX3, CAT, CASR, CASP1, CAPN1, SERPING1, BTC, HIF1A, HRG, HSF1, HSPB1, CCL3, SCT, S100A12, ROS1, RBP4, RARRES2, MOK, RAC1, PTPRC, PTH, PTEN, PSMC1, PRSS3, PROC, MAP2K3, PRKD1, PPY, CXCL12, SELE, SFTPD, PPP1R11, TYMS, TNFAIP6, TNFAIP3, TM7SF2, THPO, TFRC, TFAM, TCF3, SLC2A1, STIM1, SSRP1, SOX9, SOD1, SNTB1, SNAP25, SLC6A2, PTPA, PPIA, PNLIPRP2, IL13, KRT5, KNG1, KDR, ITPR3, ITPR1, ISG20, IL18, IL12B, LTA, IL7R, IL4, IL2RA, IFNB1, IFIT1, IAPP, HSPB2, LGALS3, SMAD2, PNLIP, NPPA, PLG, PLA2G2A, PDCD1, PAK1, SERPINE1, PAEP, NTS, NPY, SMAD7, NFKBIA, MYO9B, COX2, MMP2, MLN, MIF, MBL2, H3P45
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Hemiparesis
Wikipedia
The higher the score, the better movement and mobility is available for the individual being scored. [28] Treatment [ edit ] Treatment for hemiparesis is the same treatment given to those recovering from strokes or brain injuries. [1] Health care professionals such as physical therapists and occupational therapists play a large role in assisting these patients in their recovery.BCHE, PLAT, ATP1A2, COL4A1, SCN1A, SLC2A1, ALAD, STAT4, KLRC4, TTR, TSC2, TSC1, TPP2, TLR4, TGFBR3, SLC1A3, SMARCB1, CFHR3, RASA1, PRNP, PDGFB, OPA1, NOTCH1, DNM1L, TREX1, PDCD10, DOCK6, TUBB2B, CYP26C1, UBAC2, EOGT, WDR62, IL23R, ANGPTL6, IRF2BPL, ARHGAP31, NF2, TBC1D24, DLL4, ADA2, ERAP1, SUFU, PNPLA8, TBK1, RHOBTB2, NID1, TRNW, FAS, IL12A, TRNV, SP110, HLA-DQB1, HLA-B, CFHR1, CFH, GNAQ, GBE1, GALC, MTOR, FGFR1, ENG, COL3A1, CCR1, CACNA1A, C4A, ATP1A3, IL10, RBPJ, KRAS, ND5, TRNS2, TRNS1, TRNQ, TRNL1, TRNK, MEFV, ND6, TRNF, ND1, CYTB, COX3, COX2, COX1, TRNC, IL12A-AS1, FAP, ENPEP, MMP9, BDNF, NOTCH3, CIMT
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Inflammatory Bowel Disease
Wikipedia
Further evidence of the role of gut flora in the cause of inflammatory bowel disease is that IBD-affected individuals are more likely to have been prescribed antibiotics in the 2–5 year period before their diagnosis than unaffected individuals. [28] The enteral bacteria can be altered by environmental factors, such as concentrated milk fats (a common ingredient of processed foods and confectionery) or oral medications such as antibiotics and oral iron preparations. [29] Breach of intestinal barrier [ edit ] Further information: Intestinal permeability Loss of integrity of the intestinal epithelium plays a key pathogenic role in IBD. [30] Dysfunction of the innate immune system as a result of abnormal signaling through immune receptors called toll-like receptors (TLRs)—which activates an immune response to molecules that are broadly shared by multiple pathogens—contributes to acute and chronic inflammatory processes in IBD colitis and associated cancer. [31] Changes in the composition of the intestinal microbiota are an important environmental factor in the development of IBD. ... "Efficacy of erythropoietin combined with enteral nutrition for the treatment of anemia in Crohn's disease: a prospective cohort study". Nutrition in Clinical Practice . 28 (1): 120–7. doi : 10.1177/0884533612462744 .IL10, TNF, NOD2, CARD9, ATG16L1, IL23R, TNFSF15, IRGM, INAVA, IL18RAP, IL10RA, STAT3, PLCG2, PTPN22, ICAM1, PTGS2, TGFB1, MUC19, XIAP, IL6, APC, IL10RB, SLC11A1, IL2RA, MAGI2, DEFA5, RASSF1, ITGA4, SFRP2, APC2, VNN1, ITGAL, SFRP1, CUL2, ITGB8, HPS1, IL2, SLAMF8, XBP1, NOS2, MUC2, ESR2, RUNX3, CD40LG, NFIL3, KLKB1, HLA-DRB1, SLC6A14, IGF2, SLC22A4, F5, HLA-B, PTPN2, MST1, NUDT15, STAT4, CCL20, IL27, FUT2, TYK2, LRRK2, HLA-DQA1, TNFRSF6B, PRDM1, IL18R1, ITLN1, SMAD3, NR5A2, FCGR2A, GPR65, SLC22A23, TRAF3IP2, GPR35, CREM, LACC1, ZMIZ1, IFNG-AS1, PRKCB, SNX20, MAP3K8, CDYL2, CELSR3, NFATC1, ERAP2, ETS1, SP140, GPR183, FTO, TMBIM1, CRTC3, BSN, FERMT1, TET2, IL1RL1, IFNGR2, BANK1, ADCY7, PYGO2, CD226, SHC1, ADCY3, GALM, PLAU, S100A12, IFNG, IBD2, BACH2, ZNF831, PLA2G4A, EPHB4, CCDC26, THADA, CXCL8, PPARG, IL21, PSMG4, IL1RN, IL1B, IL1A, CDC42SE2, ERGIC1, RIT1, IL4, LINC01620, TEX41, SLAIN2, BRD2, C1orf141, EEF1AKMT2, DNMT3B, GRID2IP, IL9, RMI2, C17orf67, IL33, TSPAN14, GCKR, KIAA1109, MAML2, ZGPAT, MEFV, SMAD7, PUS10, GNA12, SLC39A11, LPP, IL12B, PLXNA4, IRF6, HGFAC, ZNF300, GPR18, LCN2, AMZ1, FAM171B, NLRP3, DENND1B, GALC, MTHFR, IPMK, DUSP16, ABCB1, IFIH1, PTK2B, DUPD1, UBAC2, ITPA, BTBD8, FEN1, NOTCH1, HMGB1, ANKRD55, NFKB1, IL17A, IL18, MYO9B, SEMA6D, CPEB4, HORMAD2, FOSL2, HLA-DQA2, NDFIP1, ATXN2, ZNF300P1, DAP, DAP3, BOK-AS1, SKAP2, NR1I2, RPSAP35, KSR1, ACAD8, SPATA48, LINC00484, CAMK2A, FOXP1, IL37, CUL1, GATD3A, CCND3, TUBD1, CD6, CD14, LINC00598, CD28, TRAF3IP2-AS1, VDR, DELEC1, UBE2L3, IL22, IMPG2, IBD5, FOXP3, MIR3936HG, SH2B1, PNKD, SPDEF, OSMR, RGS14, CIT, LINC02128, CDC37, TAB2-AS1, NOD1, ALB, GLYAT, ATXN2L, LINC02745, LINC00824, CRTC3-AS1, C5orf66, PARK7, SBNO2, SCAMP3, SH2B3, TAB2, TBC1D9, RTEL1-TNFRSF6B, TM9SF4, PHACTR2, ARSA, LITAF, FADS2, LPXN, DLG5, TPMT, SYN3, TP53, TLR2, CRP, TREM1, CDKAL1, C10orf55, SUFU, INTS11, IRF1-AS1, CTLA4, LRRC8D, CCR5, HDAC7, IL23A, TG, TIMP1, SLC22A5, TLR4, AHR, IBD3, NKX2-3, JAK2, CLDN2, TLR9, TAC1, SLC15A1, MIR155, MMP9, MIR21, COX2, DUOX2, DPP4, NOX1, IL15, HAMP, IL13, IRF5, HIF1A, CSF2, TLR5, LEP, MYDGF, F2, STAT6, IL17F, CCL2, MICA, PSC, MAPK1, HPGDS, CXCR4, MADCAM1, MPO, CYBB, GSTM1, NR3C1, TTC7A, S100A8, IGF1, BPI, NR0B1, MIR146A, GP2, IL17D, MPZ, PMP22, P2RX7, RIPK1, MBL2, AGT, MTCO2P12, IMPACT, CARD8, MIF, S100A9, LANCL1, CCR9, SIRT1, IL7, ACE, MIR223, SLC9A3, CXCL10, REG1A, RABEPK, DEFB4A, EBNA1BP2, MTFMT, HNF4A, DECR1, H3P28, MMP1, TACR1, ENTPD1, MYLK, MUC1, CD40, FCGR3A, CCR6, CAT, TP63, ARMH1, GH1, TFF3, IBD6, ARHGEF2, GUCY2C, GSTT1, LRBA, PSMD7, RPSA, KRT8, KRAS, TNFAIP6, OSM, TLR3, CCL11, SLCO6A1, AGR2, IL25, CX3CL1, VEGFA, PDCD1, ADIPOQ, WAS, NAT2, NFE2L2, RAC1, PROC, LYZ, TNFRSF25, MLH1, REG4, IL16, NLRP1, TCF4, SETD2, EBI3, PTGER4, IL11, S100B, VIP, SOAT1, MMP3, FGL2, ESR1, DEFB4B, BCL2, CD68, HGF, MTOR, FOLH1, FGF7, FCGR3B, AGER, HSPA4, DMBT1, F2RL1, EZH2, CD24, NPSR1, GSTK1, PI3, MMP2, VIPR1, CD44, MAPK8, EGF, TRAF6, GCG, CD1D, MMP13, TNFRSF1A, CEACAM5, CFTR, GBP1, TLR1, BEST1, XDH, GABPA, GJB2, IL19, AICDA, CD274, LTA, CASP3, CASP1, CXCR3, LGALS3, G6PC3, MBTPS1, CDKN2B-AS1, EGFR, TNFSF14, TIMP3, PTEN, BTNL2, SELENOP, PHB, REN, RORC, ATN1, LANCL2, DMD, PLA2G1B, MUC13, IL26, DEFB1, CCL5, SERPINE1, CXCL12, DSG2, TNFRSF11B, CHI3L1, SFTPD, NLRX1, CSF3, NM, PON1, MIR31, SOD1, GPX1, ERAP1, ADAM17, CNR1, TCF7L2, MUC5AC, TRPV4, CNR2, CUZD1, ACTB, HSPD1, DOCK11, CCND1, IL32, IL24, INSRR, IL2RB, IL4R, RBM45, RAB4B-EGLN2, MAST3, IL6ST, IRF4, NR4A1, TLR6, ITGAX, AGA, CXCR2, ISG20, PANX1, ITGAE, HLA-DPB1, EGLN2, IGFBP3, LINC02605, HMOX1, GSTM2, IFNA1, IFNA13, LINC-ROR, NR1I3, IFNGR1, CCL25, SLC52A1, BIRC3, CXCL11, CXCL5, CCN2, MIR200B, NAMPT, SDC1, CYP2D6, MIR19B1, PERCC1, AKT1, SELE, SELL, CX3CR1, SGK1, ALOX15, ALPI, MIR150, CYP3A4, FBXW7, IL1RL2, S100A4, CCL26, CXCR6, PRTN3, LILRB1, EDN1, ACACA, PTGS1, MIR122, IMMT, DUSP1, PTPN11, TNFSF13B, RAC2, ACP1, RETN, RAG2, RARRES2, SCN10A, REG1B, RELA, MIR126, RNASE3, SLC26A3, RIPK3, ADA, DNTT, ADORA2B, S100A1, MIR132, UBD, SAA1, ADORA3, MIR224, ST14, CSF1, SGSM3, CCK, TBX21, MSC, VWF, LONP1, SMIM25, MS4A1, CD207, EZR, VEGFC, VDAC1, VCAM1, SMUG1, LPAR3, ATF3, BCL3, FZD4, MAPK14, ULK1, ADAM15, HDAC3, SPHK1, BMI1, CAMP, BDNF, CLDN1, SLC16A4, TNFSF11, TCAP, IL17C, IKBKG, SLC16A3, CASP8, CASP9, ABCG2, AIM2, CDC42, HSP90B1, TFF1, GPR166P, TRBV20OR9-2, CMA1, VN1R17P, SLC23A1, TANK, SYT1, STATH, STAT1, RIPK2, CRH, SPP1, KLRK1, APOA4, MIR135B, AQP6, TFRC, TNFAIP3, MIR375, ARHGEF6, HSPA14, TNFRSF1B, TBPL1, CDKN2A, CDX2, CHGA, KLRC4-KLRK1, SATB2, CLOCK, TIMP2, TLR8, ACSBG1, ELANE, GPR151, MIR15A, PAEP, MMP14, HLA-C, RHBDF2, GPX2, MUL1, SLC11A2, MMP28, GPRC6A, MMP7, INPP5D, WNK1, IDO1, HSD11B1, PAK1, REG3A, PRRT2, GDNF, FFAR2, GEM, CELIAC2, PDE4A, IL13RA2, HP, GORASP1, GPBAR1, NOS3, MRGPRX4, ITGAM, KIR3DL1, KIR2DS1, MUC6, MUC4, MRGPRX1, CEACAM6, OXER1, JAK3, FN1, EGLN3, FPR2, CBLL2, NCF4, GAST, NEAT1, FHIT, GAPDH, IL22RA2, GC, MRGPRX3, MOG, NGF, KIF22, CLEC7A, PRKN, GJA1, HT, IGAN1, IL1F10, PKM, GPR3, LINC01193, MEP1A, PLG, HSP90AA1, IL6R, POMC, PIM1, PON2, LGR6, IL1R1, IRF8, IDH1, GLI1, GLB1, PRKCA, EPAS1, PIK3CD, MYD88, HSD11B2, CBLIF, GAS5, LGALS9, HSPA1A, MICB, F3, HSPA1B, F2R, C20orf181, SLC36A1, CHMP2B, MIR1260A, MYLK3, REXO2, MIR1246, RABGEF1, POLDIP2, GGTLC1, MIR3194, MCAT, IL36A, TIGIT, RNF19A, SUMF2, TSLP, TRIB2, PART1, CAVIN3, GGTLC3, MCTS1, MUC5B, CCR2, ATG16L2, GGT2, IBD8, IBD27, MUC16, ORMDL3, IBD20, MIR665, IBD15, MLKL, IL17B, DNAI1, VENTX, RNU1-1, PGAP3, SND1, REEP6, ADAMDEC1, B3GAT1, IBD21, ASXL1, IL36RN, TMEM201, NLRP12, NLRP6, FGF20, PHGDH, GGTLC4P, H3C9P, CPAMD8, DAB2IP, MIR4284, WIF1, SLC2A14, AKAP13, HPS5, ERVK-19, PACSIN2, ZPBP2, CBSL, LOC102724197, NRG4, SCN11A, ERVK-24, CBX3, CDAN1, ERVK-25, ZFP90, ERVK-18, ERVK-21, IRAK3, WBP4, LRRC3C, OR2AG1, TAAR1, EHD1, RNF183, SCLT1, STIN2-VNTR, RAB32, ACTRT1, SDS, MUC17, LILRB4, NEK7, SGPP2, ADAMTS13, EMILIN1, CD160, LAYN, WDHD1, CARMIL2, ATF6, IL34, DKK1, CBR3-AS1, ZNF281, SEC14L2, AGR3, PADI4, FCRL3, MYSM1, AMACR, LY96, PLA2G15, TMEFF2, TFIP11, TIRAP, IFNLR1, CLDN15, DBA2, SLC52A3, MIR595, CABIN1, SLC39A14, NXPE1, ERVK-9, SLC15A4, KIF21B, ERVK-10, FAM24B-CUZD1, COMMD1, MPRIP, ARL6IP1, LRG1, IBD11, MLC1, FKBP15, CCDC80, OCLN, TPSD1, SF3B1, C1GALT1C1, CCL4L1, H3P37, MIR18A, ATG9A, MIR192, UBE2Z, BATF3, KIF9-AS1, RAPH1, MIR185, NDRG4, BCL11B, USE1, AMBRA1, ATF7IP, MIR149, IFT122, FNDC4, PPP1R42, MIR148A, HCAR2, SLC9C1, CARD14, QRSL1, MIR196A2, RNF186, EGLN1, UGT1A1, MIR301A, MIR29A, PGP, AFTPH, MS4A12, MORC4, GSDMA, MIR22, MIR204, MARCHF1, CXCL17, STEAP4, ANKZF1, PBRM1, SELENOS, MIR142, CCDC88B, LINC01194, MIR124-1, TEPP, MIR10A, IBD7, IL31, RTN4, MIR106A, PRM3, SCAF1, GSDMB, KIR2DL5A, SENP7, CXCL16, MUC3B, TAOK1, ZNF410, MRTFA, PRX, FAM20C, DUSP22, MIR125A, SLC25A19, MIR141, DCLRE1C, FAM92B, DEFB103B, ACSS2, DIO3OS, NSFL1C, SLC39A8, SLC28A3, SLC6A19, STING1, PROK2, PARD3, ASAH2, TRIM39, RSPO2, DNAAF3, SMOX, KRT20, PYCARD, F11R, SCART1, CARD11, NPS, MIR505, IL20, IBD4, IL21R, MIR495, MIR146B, ERVK-7, MAGT1, KIR2DL5B, JAZF1, MALT1, SOST, EXOSC1, ADIPOR1, MIR425, FGD2, IL17REL, CRNDE, LTO1, CLEC2D, MYLK2, UHRF1, USP25, SNX10, GGTLC5P, SHANK3, CERS2, MDFIC, PILRA, RSPO3, SFTPA1, HAVCR2, NCF1, CD209, ACCS, TRIM63, EIF2A, BCL2L12, TOLLIP, ELOVL7, GDE1, KLF13, DEFB103A, MAGI3, IFNL2, MINDY3, MIR7-3, DUOX1, MIR7-2, ARHGAP24, MIR7-1, EIM, MIR34C, MIR320A, GSDMD, TREM2, SNIP1, TERF2IP, PIM3, FLAD1, TAC4, SCLY, VMP1, MIR374A, MAP1LC3B, MIR346, UNC5CL, NT5C3A, SGPP1, MIR148B, TLR7, NPL, TNFRSF12A, CRNKL1, PNPLA3, POP5, PPP1R2C, COMMD10, ISYNA1, ERVK-8, NAT1, HPSE, HLA-A, GPR4, GPER1, GPT, GRN, GTF2F1, GUCA2A, GUCA2B, GUSB, GZMB, GZMM, HADHA, HDAC2, HFE, HGD, HLA-DOA, GNAI2, HLA-DPA1, HLA-DQB1, HLA-DQB2, HLA-DRA, HLA-DRB5, HLA-G, SLC29A2, HPRT1, ERAS, HSPA1L, HSPA2, HSPB1, HSPB2, TNC, GNAO1, GLP1R, IQGAP2, FGB, ERF, ERN1, ESRRB, ETV5, ACSL1, FASN, FBN2, FCER2, FCGR1A, FCGR1B, FCGRT, FCN1, FCN2, FER, FGF2, GLI3, FGFR1, FGFR2, FOXO1, FOXO3, FLG, FLNB, FLT3LG, FTL, ACKR1, GATA3, KAT2A, GDF1, GGT1, GHSR, IAPP, ICA1, ICAM3, SMAD4, KNG1, KRT1, KRT7, KRT18, L1CAM, LGALS1, LGALS2, LGALS3BP, LIF, LMNA, LPL, LTA4H, LTB, SH2D1A, SMAD5, IDH2, MAN2A1, MCC, MDK, MGAT1, MGAT3, MGMT, CIITA, CD99, MIP, MLN, FOXO4, MMP8, MMP10, MMP12, KLK1, KIR3DS1, KIR3DL2, KIR2DS5, IDUA, IFI16, IFN1@, IFNAR1, IFNB1, IGF1R, IGFBP4, IKBKB, IL3, IL5, CXCR1, IL12A, IL12RB1, IL15RA, IMPDH1, ING1, IREB2, IRF1, ITGA2B, ITGB1, ITGB2, ITGB3, ITIH1, ITIH2, ITIH3, JAK1, KCNN4, KCNQ2, KIR2DL2, ERBB4, ERBB2, EPO, CALCA, CFB, BMP1, BMP2, BMP7, BRCA1, BRAF, BTG1, TSPO, C3, C4B, VPS51, CA2, SLC25A20, CAD, CALCR, TNFSF8, CALR, CAMK4, CAST, CASP7, CASR, CBFA2T3, CBR1, CBS, KYAT1, CCNE1, CD3G, CD19, CD80, CD86, BDKRB2, TNFRSF17, AVP, ATF4, AOC1, ACHE, ADCYAP1, ADM, AFP, AHSG, AIF1, ALCAM, ALOX5, ALPP, AMCN, ANGPT2, ANXA1, ANXA2, AIRE, BIRC2, APOA1, AQP8, APOE, KLK3, FAS, AQP3, AQP5, AQP9, AREG, ARRB2, STS, ARSF, ASCL2, TNFRSF8, CD36, STX2, HBEGF, CYP27B1, CD55, DBH, DBP, DCC, DDX3X, DEFA6, DGUOK, DLD, DMRT1, DNAH5, DNMT1, DRD2, TSC22D3, DVL1, CD38, ECM1, S1PR1, EEF1A1, EGR1, ELF1, ELK3, ELK4, CTTN, ENO1, SLC29A1, EPHB1, EPHB2, EPHX1, EPHX2, CUX1, CTSG, CTNNB1, CTF1, CD47, CD59, CD70, CDH1, CDK9, CDKN1A, CDKN1B, CDKN2B, CES1, CGA, CHRNA5, CLCN5, CCR3, LTB4R, CMM, CNGA1, COL1A1, KLF6, COX8A, CLDN4, CPOX, CPT1A, CRK, CRX, CRYAB, CSF1R, CSF2RA, CSF3R, VCAN, MMP19, MNAT1, MPG, PIR, FOSL1, GPR68, SLC7A5, CDR3, MIA, NRIP1, BAS, AXIN1, EOMES, PLA2G6, STK24, DHX16, PPM1D, HAT1, PIAS1, PHOX2B, USO1, RNASET2, HYAL2, S1PR4, TNK1, TNFRSF14, CES2, GMPS, SOCS2, CCN4, KAT2B, APLN, PER3, PER2, CCDC6, AIMP2, LST1, ZNF133, TFF2, TGFA, TGFB2, TGFBR2, TIAL1, TJP1, TP73, TPH1, TRAF1, TRAF2, TRAF3, TRAF5, TST, TUFM, TNFSF4, TNFRSF4, TXK, TYROBP, TYRP1, UBC, UBE2I, UCP2, VCL, VIM, TRPV1, WARS1, WNT1, YWHAE, ZFP36, MTMR3, HSPB3, TERF2, NDRG1, HDAC6, PDCD6IP, IL18BP, PTPRU, ARPC2, LRPPRC, OPTN, ABI2, SLC25A15, POP7, ABCC4, IGSF6, TNIP1, TRIM22, RACK1, PGLYRP1, FST, NXF1, APPBP2, FBLN5, IPO8, OLFM4, SCGN, AHSA1, USP16, PDPN, POSTN, AVIL, CORIN, CERS1, NR1H4, USP15, WDR1, FCHSD2, NOL3, SOCS3, PSTPIP1, CLDN8, LATS1, MTA2, MAGI1, GPR55, S1PR2, CD163, SLIT2, SLC9A3R1, KIF3B, PLAA, COX5A, CD101, GRAP2, NTN1, GSTO1, EIF2AK3, ROCK2, PTGES, CXCL14, CCL4L2, PDIA4, SOCS5, HDAC4, ZEB2, ZBTB24, TERT, TEK, MRC1, PLAT, PDZK1, PECAM1, PEPD, PF4, CFP, PGA5, PGGT1B, SLC25A3, SERPINE2, PIGA, PIK3C3, PIK3CA, PIK3CB, PIK3CG, PLEK, PSMA7, PON3, POU2F1, PPARA, PPIA, PRF1, PRKAA1, PRKAA2, PRKAB1, MAPK3, EIF2AK2, PRNP, PSPN, HTRA1, PSAP, PDYN, PDGFB, PDC, PCBD1, MSD, COX1, MTR, RNR1, MTX1, MUC3A, MUSK, MYC, MYH10, NCAM1, NCF2, NEDD8, NELL1, NFKBIA, NOS1, NPY, NPR1, NPR3, NPY2R, NTRK1, NTS, NTSR1, ODC1, OPRM1, P2RX5, P4HB, PAFAH1B1, PAPPA, PC, PSG5, PSMD4, TCOF1, SUMO2, SLC2A3, SLC2A5, SLC3A2, SLC6A2, SLC6A4, SLC6A9, SLC7A2, SLC9A1, SLC9A2, SLC16A1, SLCO2A1, SLC22A1, SMS, SUMO3, SNCA, PSMD10, SOD3, TRIM21, RO60, SST, ST2, STAT5A, STAT5B, SULT1E1, SYK, TACR2, TAL1, TBP, TCEA1, TCF21, SI, SHBG, SGCG, SRSF1, PTAFR, PTCH1, PTGDS, PTPRC, PTPRH, PTPRS, PTPRZ1, MOK, RAP1A, RAPSN, RBM3, REL, RNASE2, RNF5, RNU1-4, ROS1, RPLP0, RPLP2, S100A10, SAA2, SAA3P, CLEC11A, CCL4, CCL7, CCL14, CCL15, CCL17, CCL22, SELP, PRDX2
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Fish Disease And Parasites
Wikipedia
These small fish maintain so-called "cleaning stations" where other fish, known as hosts, will congregate and perform specific movements to attract the attention of the cleaner fish. [27] Cleaning behaviours have been observed in a number of other fish groups, including an interesting case between two cichlids of the same genus, Etroplus maculatus , the cleaner fish, and the much larger Etroplus suratensis , the host. [28] More than 40 species of parasites may reside on the skin and internally of the ocean sunfish , motivating the fish to seek relief in a number of ways. [29] [30] In temperate regions, drifting kelp fields harbour cleaner wrasses and other fish which remove parasites from the skin of visiting sunfish. ... North American Journal of Fisheries Management . 28 (2): 523–532. doi : 10.1577/m07-042.1 . ^ Krkošek, M.; Lewis, M.A.; Morton, A.; Frazer, L.N.; Volpe, J.P. (2006).
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Mind-Blindness
Wikipedia
In fact, a survey completed in 2004 revealed that 44% of primary care pediatricians reported that they care for at least 10 children with ASDs”. [27] Relationship to schizophrenia [ edit ] People with schizophrenia also show deficits associated with mind-blindness. [3] In fact, as stated by Brune, “there is good empirical evidence that the theory of minds is specifically impaired in schizophrenia and that many psychotic symptoms—for instance, delusions of alien control and persecution, the presence of thought and language disorganization, and other behavioral symptoms—may best be understood in light of a disturbed capacity in patients to relate their own intentions to executing behavior, and to monitor others’ intentions.” [28] However, there is an ongoing debate as to whether individuals with schizophrenia have an impaired ToM leading to mind-blindness or display an exaggerated ToM.
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Subarachnoid Hemorrhage
Wikipedia
If the number of cells decreases per bottle, it is more likely that it is due to damage to a small blood vessel during the procedure (known as a "traumatic tap"). [24] While there is no official cutoff for red blood cells in the CSF no documented cases have occurred at less than "a few hundred cells" per high-powered field. [28] The CSF sample is also examined for xanthochromia —the yellow appearance of centrifugated fluid. ... "Traumatic subarachnoid hemorrhage: our current understanding and its evolution over the past half century". Neurological Research . 28 (4): 445–52. doi : 10.1179/016164106X115053 . ... "Surgical risk as related to time of intervention in the repair of intracranial aneurysms" . Journal of Neurosurgery . 28 (1): 14–20. doi : 10.3171/jns.1968.28.1.0014 .CASP3, EDN1, PPARG, NTN1, THSD1, UNC5B, NOS2, EDNRB, NOS3, ADORA1, MMP9, TLR4, TNF, BCL2, TNC, NFE2L2, HMOX1, AGER, BECN1, CYBB, CASP9, ABCC8, TRPM4, ICAM1, STAT3, GSK3B, NGB, TJP1, MMP2, NTRK2, HTR1B, HSPD1, MMP13, GLI1, TACR1, SHH, KCNJ10, NQO1, PRKN, ESR1, PTCH1, NLK, CCL20, HIF1A, CXCL1, BNIP3, HCN1, C3, ELN, ENG, ITGB3, COL3A1, MYLK, PRKG1, TGFB2, PIK3CB, PIK3CA, F10, FBN1, TGFBR1, ACSM3, TGFB3, MFAP5, TGFBR2, TGFBR3, ACTA2, ACVRL1, CD109, NLRP3, ANGPTL6, DOCK8, HP, HMGB1, APOE, IL6, ITGA2, ITGA2B, GP1BB, GP1BA, LAMC2, LOX, SMAD3, SMAD4, GDF2, PIK3CD, MAT2A, FOXE3, MYH11, CRP, CSF2, PIK3CG, IL1B, MAPK1, SIRT1, BDNF, ALB, ACE, COL4A2, MPO, SPP1, CEBPZ, RIPK3, MAPK8, AQP4, VEGFA, EPHB2, POSTN, MAPK14, S100B, TXNIP, OR10A4, XYLT2, LGALS3, CALCA, F3, ADAMTS13, IL1A, TP53, MPRIP, GABPA, SOD1, JAK2, MST1, IL10, POLDIP2, SIRT3, TREM1, RNF19A, APLN, AHSA1, DNM1L, GRAP2, RIPK1, AIMP2, MIF, VWF, VCAM1, MYD88, UGCG, SERPINE1, S100A12, RYR1, PDGFRB, REN, SOD2, SERPINA3, AVP, GCLC, AGTR1, EPO, CBS, CRK, GRM1, GFAP, AKT1, ROCK2, AGT, TMBIM6, CTNNB1, SARDH, CX3CR1, MTOR, PKD1, ATF4, CASP1, MOK, ERBB4, CD163, TBL1X, P2RX7, HAMP, NPY, SORD, FCN1, AIF1, NEFL, PTK2B, UTS2, PCSK1, RALBP1, SDS, DAPK3, F2, SDHB, PHB2, DDIT3, IL17A, ENO2, DAPK2, EGFR, SMUG1, DENR, VDR, MAP2K1, MIR502, MIR30A, OCLN, UTRN, UQCRFS1, CBSL, TTR, PRKAB1, PRKAA2, HSPB3, ABO, GLP1R, PRKAA1, TNFRSF1A, CRMP1, CLDN5, AQP1, C20orf181, MASP2, NGEF, PANX1, BIRC2, KDM6B, HPSE, TARDBP, BGN, XIAP, APP, ATF6, MAPK8IP3, HSP90AA1, TSPO, ERLIN1, HAP1, SELENBP1, DLK1, TNFSF12, CD36, MBTPS1, S1PR4, PEA15, CD38, FCN3, PLA2G6, ST8SIA2, CD44, CD68, PLA2G7, RPS6KA5, KLF4, ADIPOQ, C5AR1, C5, YAP1, IRF9, APBB3, NET1, SLC25A15, CAST, CASP8, APPL1, KEAP1, PCLAF, HDAC4, CLOCK, ATG5, SRA1, SIRT6, ANXA7, SLA2, CPP, TSPAN33, SCARA5, BTBD8, ADM, COPD, ADRB1, MLKL, GPBAR1, LYPD4, ROMO1, ADRB2, HSPB6, NEXN, IL33, RIOX2, ZGPAT, MIR132, MIR143, MIR145, MIR532, RN7SL263P, MIR4793, MIR1297, H3P36, MIR675, CCR2, MIR630, ECSCR, MIR15A, MIR324, MIR24-1, MIR221, MIR22, MIR21, MIR206, MIR183, POLDIP3, RAB34, CLEC4E, TRAPPC9, MOCOS, ALDH2, PDP1, DDIT4, CCHCR1, TERF2IP, ALOX15, ANGPT1, CXCR4, PRRX2, NRN1, TLR7, NOX4, SETD2, SGSM3, RABGEF1, INTU, AGGF1, PPP6R3, MEG3, WNK1, APLNR, ITM2C, PPP1R2C, RIOX1, MMEL1, MAPKAP1, WNK2, PINK1, AKT2, GORASP1, SOX17, RXFP1, ACE2, MRS2, PELI1, ZC4H2, MANF, TRAF2, MAP3K12, FPR2, MTHFR, FGFR3, MSR1, MRC1, FOXO3, MPG, MNT, MMP12, FGFR1, MMP10, MMP7, MMP3, NR5A2, MMP1, CXCL9, GAP43, MYC, FGF13, PLAT, OLR1, PLA2G1B, ERN1, F2R, PDE4A, PCMT1, FCGR3A, PRDX1, OCA2, FGF2, NUCB2, FCGR3B, NPPB, NOTCH1, FDPS, FES, NEFH, MET, MAP3K5, MEFV, HLA-A, IL18, CXCL3, CXCL8, IL7, IL6R, NRG1, IL1RN, HPX, SMCP, CCN1, IGF1, IFNA13, IFNA1, HSPA4, HSPA5, HSPB1, CXCL10, IRAK1, GRIN2B, GRIA1, MC1R, MBP, KAT2A, MAS1, MARS1, MAP2, GJA1, LRPAP1, LIMK1, LCN2, GLS, LAMP1, GPER1, GPT, ITPR3, PLA2G2A, PLCD1, SLC30A3, CTSG, TLR2, CRYGD, TIMP3, TIMP1, THBD, TH, CCN2, CYP3A4, CRY2, TFPI, PRDX2, TAT, MAP3K7, SYT1, SYN1, DBH, TLR3, TM7SF2, PLXNB1, CLU, ZMYM2, ZFP36, WNT1, CEBPA, VTN, VIP, CHRM3, VASP, TNNI3, CLDN3, UCP3, UCP2, TRH, TRAF6, HSPB2, NR2C2, DBP, SSRP1, SPTAN1, PTEN, EGF, RELA, RAP1A, PTK2, PTGS2, PTGS1, PTGER4, MASP1, ECI1, MAP2K7, EGR1, MAPK3, ABCA1, PTPA, EPHA4, PRRX1, TRIM27, BRD2, RNPEP, ROCK1, DCX, SLC9A1, SLC2A1, DECR1, SHBG, SELENOP, SELP, CCL13, CCL2, HBEGF, E2F1, S1PR1, S100A1, EDNRA, ROS1, LINC02605
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Scoliosis
Wikipedia
Some conditions which may cause secondary scoliosis include muscular dystrophy , spinal muscular atrophy , poliomyelitis , cerebral palsy , spinal cord trauma, and myotonia . [28] [29] Scoliosis often presents itself, or worsens, during an adolescent's growth spurt and is more often diagnosed in females than males. [25] Scoliosis associated with known syndromes is often subclassified as "syndromic scoliosis". [ citation needed ] Scoliosis can be associated with amniotic band syndrome , Arnold–Chiari malformation , Charcot–Marie–Tooth disease , cerebral palsy , congenital diaphragmatic hernia , connective tissue disorders , muscular dystrophy , familial dysautonomia , CHARGE syndrome , Ehlers–Danlos syndrome (hyperflexibility, "floppy baby" syndrome, and other variants of the condition), fragile X syndrome , [30] [31] Friedreich's ataxia , hemihypertrophy , Loeys–Dietz syndrome , Marfan syndrome , nail–patella syndrome , neurofibromatosis , osteogenesis imperfecta , Prader–Willi syndrome , proteus syndrome , spina bifida , spinal muscular atrophy , syringomyelia , and pectus carinatum . [ citation needed ] Another form of secondary scoliosis is degenerative scoliosis, also known as de novo scoliosis, which develops later in life secondary to degenerative (may or may not be associated with aging) changes. ... ISBN 978-1439871744 . [ page needed ] ^ Health and Function of Patients With Untreated Idiopathic Scoliosis – Reply [1] ^ Many With Scoliosis Can Skip Treatments Archived 20 August 2008 at the Wayback Machine ^ "Adolescent Idiopathic Scoliosis | Scoliosis Research Society" . www.srs.org . Retrieved 28 February 2018 . ^ Edery P, Margaritte-Jeannin P, Biot B, Labalme A, Bernard JC, Chastang J, Kassai B, Plais MH, Moldovan F, Clerget-Darpoux F (August 2011).DLL3, ATAD3A, DLL1, SLC10A7, PIGU, PAX7, NOTCH1, TRAPPC4, FGFR3, MMP1, ROBO3, SH3TC2, PIEZO2, MECP2, TGFB1, COL1A1, FXN, STAT3, SELENON, FLNB, PMP22, EXT1, LBX1, RET, COL5A1, RUNX2, PLOD1, COL3A1, COL1A2, GJC2, SOX9, NGLY1, SOX5, SMAD3, ELN, FBN1, NUS1, SHOX, CCN6, RIN2, MYF5, PTPN11, SLC26A2, PTCH1, CHD7, CREBBP, CRLF1, NRXN1, ZMYND10, RYR1, LOX, ZBTB20, DNAH1, AUTS2, PHGDH, CNTNAP2, B3GAT3, ABCB7, LEMD3, SH2B1, SUZ12, KIAA0556, BICD2, RPGRIP1L, NEDD4L, SYNE1, OBSL1, SLC35A3, TGDS, SLC39A14, SIN3A, KAT6B, CLCF1, ATP6V0A2, LRRC6, NPAP1, FLRT1, RAB3GAP2, SAMHD1, TINF2, SETD2, HSPB8, OTUD6B, ZDHHC9, SBDS, TMEM216, NME8, DACT1, MBTPS2, TMEM138, LARP7, PIGT, SUFU, BSCL2, RAB23, RTEL1, INPP5K, MAP3K20, FGFRL1, GDAP1, TMCO1, MAGEL2, GTPBP2, TRAPPC12, NDUFAF1, YARS2, ATL1, TBL2, DNAI1, B9D1, STK36, TUBGCP4, PGAP2, CCDC22, FLVCR1, UBE2T, ANKRD11, STRN4, GMPPB, POMT2, SLC25A24, PSAT1, RRM2B, CHST11, NSDHL, CDON, EXOC6B, RAB18, SYNE2, CEP104, PIGL, TBX4, ADAMTS2, BAG3, GABBR2, GTF2IRD1, GOSR2, GDF3, SEC24C, SEMA3E, KMT2B, ECEL1, TMEM94, PIEZO1, KIAA0586, PTDSS1, CUL7, DNAJC6, RUSC2, SEC24D, WASHC5, MFN2, CHST3, CYP7B1, MED12, HERC1, DNAH11, EED, BANF1, DPM2, SYNJ1, MCM3AP, MTMR2, AP1S2, CACNA1G, HERC2, FOXH1, RECQL4, PLOD3, BAZ1B, SEMA5A, AIFM1, CTDP1, LARGE1, TRIP11, TRIP4, LONP1, COG1, RBM8A, SLC12A6, PMPCA, MRAS, RAI1, CPLX1, KCNQ1OT1, TLK2, PRDM5, POLR3A, ERLIN2, PDCD10, TREX1, RNF13, PUF60, PLK4, RAB3GAP1, HYDIN, DKK1, CEP152, ZNF292, EMC1, ZNF423, IQSEC2, PHF8, MAPK8IP3, STAG2, EBP, GNE, MAD2L2, HUWE1, PQBP1, MKRN3-AS1, ATP6AP2, CD96, SF3B4, TRAIP, APC2, CCNO, KLHL41, PIBF1, POMT1, SLC9A6, SEC23B, SEC23A, CRTAP, FBLN5, DEAF1, RNASEH2A, AGPAT2, ARFGEF2, COLEC10, P4HTM, WRAP53, AHI1, LRRC56, NEK9, GORAB, DRC1, MARS2, PGAP3, CCDC114, CEP41, NACC1, CHST14, SLC52A3, CCDC151, RSS, SP7, FGD4, DNAAF1, CANT1, B3GALT6, TBC1D20, DNAJC21, RIPPLY2, NDUFAF6, PIH1D3, TMEM67, SLC25A46, KLC4, RSPH1, ANTXR1, POMK, PHF6, COG8, SLX4, MEGF10, NKX6-2, GNPTG, ZNF469, MYPN, HES7, FAM126A, PIGO, LMNB2, DISP1, PIGY, CFAP300, COL27A1, ALG2, TUBGCP6, CCDC65, AMER1, A2ML1, DYM, CCDC103, PIGW, CAVIN1, DOK7, SH3PXD2B, SNORD115-1, CCDC39, MCIDAS, RSPH4A, DNAAF3, DNAJB13, MYMK, B3GLCT, KBTBD13, GDF6, LBX1-AS1, ALG11, PWRN1, SNORD116-1, KLLN, TTN-AS1, LINC02294, CHL1-AS2, KANSL1, H19, NALCN, RNASEH1, PWAR1, MESP2, CCBE1, ARL6IP6, CEP120, VPS13B, FAM120AOS, TMTC3, DNAAF4, SGMS2, ARX, ASXL1, ARID2, ALG14, ARL13B, FLCN, ANO5, HYLS1, JMJD1C, RSPH9, GAS2L2, RNASEH2C, ATRIP, BRIP1, SALL4, IARS2, ERMARD, CENPJ, ZC4H2, NDUFA12, ALG1, LMOD3, INPP5E, CFAP298, RARS2, ADGRG6, CCDC8, PLEKHG5, SHROOM4, ARID1B, HACE1, CC2D2A, ALS2, FANCM, GBA2, PRX, SPTBN4, PACS1, NHP2, PIGV, FOXRED1, SAMD9, APTX, BCOR, NSUN2, NCAPG2, MKS1, DNAAF5, RNF125, CCDC40, FANCL, ARMC4, DCHS1, MSTO1, RFWD3, DNAAF2, SETD5, FANCI, ASXL2, VPS53, UFSP2, NOP10, PRUNE1, TRPV4, SLC5A7, ROGDI, DYNC2H1, TBL1XR1, PALB2, TTC21B, CSPP1, TCTN2, CTC1, CEP290, FUZ, ARMC9, PUS1, ASXL3, DDHD1, SLC2A10, ADAMTS10, VANGL1, DDX59, TTC25, DNAL1, CCM2, RSPH3, USB1, FAT4, FKBP10, EFL1, TRAPPC11, PRDM16, XYLT1, IFIH1, P3H1, NSD1, NXN, SIL1, DNAI2, KLC2, PORCN, TMEM237, UPF3B, CPLANE1, COLEC11, FKRP, GNPTAB, TMEM43, TMEM231, TCTN1, RNASEH2B, PTCH2, GNPAT, MPZ, HPGD, GRIN1, GRIN2B, GTF2I, BRF1, GUSB, HTT, HNRNPH2, HNRNPK, HNRNPU, HRAS, FLNA, HSD17B4, HSPD1, HSPG2, IDH1, IDH2, IDUA, IGBP1, IGF2, IHH, GRIA3, GP1BB, GNAQ, GLI3, FN1, FOS, FUCA1, FZD2, GABRD, GAD1, B4GALNT1, GALNS, GARS1, GAS1, GAS8, GCH1, GFAP, GFPT1, GJB1, GPC3, GLB1, GLE1, GLI2, INPPL1, IPW, IRF6, MMP13, MPV17, CDK13, MRE11, MTM1, MUSK, MYH3, MYH7, MYH11, MYLK, MYOD1, NDN, NDP, NEB, NEU1, NFIX, TONSL, NODAL, NONO, NOTCH2, MPL, MEOX1, ITGA7, MEIS2, ITGB6, KCNH1, KCNJ2, KCNJ6, KIF5A, KIF22, KRAS, LAMA2, LAMB2, LETM1, LFNG, LIFR, LIG4, LIMK1, LMNA, LMX1B, LRP4, MAP1B, MAT2A, FMR1, FLII, NPHP1, SERPINH1, KIF1A, BDNF, BMPR1B, BRCA1, BRAF, BRCA2, CACNA1C, CAPN1, CAV1, CBS, FLI1, KRIT1, CDC42, CENPE, CFL2, CHRM3, CHRNA1, CHRNG, CLCN4, COL2A1, ATR, ATP7A, RERE, ASAH1, ACADS, ACTA1, ACTA2, ACTB, ACTG1, ACVR1, ADAR, AGA, AHSG, AKT1, ABCD1, ALDH3A2, ALX3, BIN1, APC, ABCC6, ARF1, ARL3, ARVCF, COL5A2, COL6A1, COL6A2, ERCC4, EZH2, FANCA, FANCC, FANCD2, FANCE, BPTF, FANCB, FANCF, FANCG, FKTN, GPC4, FGD1, FGF8, FGF14, FGFR1, FGFR2, FHL1, FOXG1, FOXE3, EXT2, EP300, COL6A3, EMD, COL12A1, COMP, COMT, CTBP1, CTNND2, CTSK, DCC, DDX3X, DHCR7, DKC1, DMD, DNAH5, DPAGT1, DPP6, ATN1, DVL1, DVL3, TOR1A, EFNB1, NOTCH3, EMC1-AS1, NPR2, TERT, TBCD, TBCE, TBX2, TBX5, TBX6, TCF4, TCF20, TDGF1, TERC, TGFB2, TRPS1, TGFB3, TGFBR1, TGFBR2, TGIF1, TK2, TNNI2, TNNT3, TPM2, TPM3, TBX1, MAP3K7, SYT1, VAMP1, SMARCA4, SMARCB1, SMARCC2, SMARCE1, SMS, SNRPB, SNRPN, SON, SOS1, SOS2, SOX4, SOX11, SPAG1, SPARC, NRAS, SRP54, SRY, SSR4, CDKL5, TRIO, TTN, SLCO2A1, TRRAP, GAN, GDF5, LZTR1, USP9X, RBM10, NAA10, LAGE3, ARID1A, COLQ, CDC45, TTPA, LTBP4, ABL1, CUL4B, OFD1, KCNAB2, IKBKG, ELP1, DEGS1, CASK, HMGA2, KMT2D, MFAP5, ALMS1, HIRA, TWIST1, UBA1, UBE3A, UFD1, SLC35A2, KDM6A, VDR, CLIP2, WHCR, NSD2, NELFA, WNT1, WNT5A, WT1, XRCC2, YWHAG, ZIC2, MKRN3, SMARCA2, SPG7, RAP1A, RAC1, PCNT, MASP1, PSMD12, PAX6, PTEN, PYCR1, ALDH18A1, RAD51, PCYT1A, RAD51C, RAF1, PIK3C2A, PAX3, PARN, RAP1B, RAPSN, PRPS1, PDE4D, RBBP8, PEX6, PLOD2, PODXL, POLA1, POLD1, PGM3, POLE, POR, PPARG, ENPP1, PPIB, PRKAR1A, PRKD1, PRKG1, MAP2K1, MAP2K2, SLC16A2, RASA2, PIK3CA, DPF2, TRAPPC2, SDHC, ROR2, SFRP4, SGCG, SDHB, DDR2, SHH, SBF1, SIX3, RREB1, RRAS, NUP88, SKI, RPS6KA3, RPL11, RPL10, RPGR, SLC6A3, RMRP, RIT1, OCRL, RFC2, P4HB, SDHD, IS1, GH1, TP63, AR, EOS, IH, LRP2, MCF2L, FBN2, MMP3, ACAN, ATP2A2, SERPINA1, BFSP2, MT1A, ATP2B4, KIF6, ATP2A3, ACR, TNFSF11, TUBB2B, ACTN3, MTNR1A, NTF3, PLF, KRT8P3, UBL4A, DYSF, LINC00649, NTRK1, GATA6, BGLAP, CCL2, EGR2, ESR1, F9, HTC2, EFEMP1, HGF, FGF2, CX3CL1, FGF23, SPAST, FOXC2, SGCA, SATB2, SMN2, SMN1, GHR, IL1B, IL6, TPH1, TAS2R38, CCDC6, TUBA1A, CAPN3, RUNX1, ZIC1, CD38, MATN1, CHD2, CHI3L1, LEP, ASH1L, PRL, CRP, CSF1R, KRT8, REM1
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Erythromelalgia
Wikipedia
These differences in excitability alterations between the sympathetic nervous system and nociceptors is due to different expression of sodium channels other than Na V 1.7 in them. [9] What causes epidemic erythromelalgia in southern China remains unknown although several erythromelalgia-associated poxviruses were isolated from throat swabs of several patients at different counties and two different seasons. [22] [23] [24] Side effect of medication [ edit ] Several medications, including verapamil and nifedipine , as well as ergot derivatives such as bromocriptine and pergolide , have been associated with medication-induced erythromelalgia. [ citation needed ] Mushroom poisoning [ edit ] The consumption of two species of related fungi, Clitocybe acromelalga from Japan, [25] and Clitocybe amoenolens from France, [26] has led to several cases of mushroom-induced erythromelalgia which lasted from 8 days to 5 months. [27] Possible infectious cause [ edit ] An epidemic form of this syndrome occurs in secondary students in rural China. [13] In 1987 a virus - erythromelalgia-associated poxvirus - was reported to have been recovered from throat swabs from such an outbreak. [22] The genome of this virus has been sequenced and it appears that this virus is a strain of mousepox . [28] Since this virus has not yet been isolated from other outbreaks in other parts of southern China to date this putative association should be treated with circumspection. [ citation needed ] Pathophysiology [ edit ] N.B. ... "Nav1.7-A1632G Mutation from a Family with Inherited Erythromelalgia: Enhanced Firing of Dorsal Root Ganglia Neurons Evoked by Thermal Stimuli" . Journal of Neuroscience . 36 (28): 7511–7522. doi : 10.1523/JNEUROSCI.0462-16.2016 .
- Congenital Adrenal Hyperplasia Wikipedia
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Social Anxiety Disorder
Wikipedia
., avoidance of participating in small groups, avoidance of going to a party) are more likely to trigger comorbid depressive symptoms than other social fears, and thus deserve a very careful audit during clinical assessment among patients with SAD. [25] Anxiety disorders other than SAD are also very common in patients with SAD, in particular generalized anxiety disorder . [26] [27] Avoidant personality disorder is likewise highly correlated with SAD, with comorbidity rates ranging from 25% to 89%. [22] [28] [29] To try to reduce their anxiety and alleviate depression, people with social phobia may use alcohol or other drugs, which can lead to substance abuse .
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Kleptomania
Wikipedia
Psychoanalysts define the condition as an indication of a defense mechanism deriving in the unconscious ego against anxiety, prohibited intuition or desires, unsettled struggle or forbidden sexual drives, dread of castration, sexual excitement, and sexual fulfillment and orgasm throughout the act of stealing. [28] The psychoanalytic and psycho-dynamic approach to kleptomania granted the basis for prolonged psychoanalytic or psycho-dynamic psychotherapy as the core treatment method for a number of years.
