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Intrauterine Hypoxia Wikipedia

PMC 2963133 . PMID 20981293 . ^ Mund M, Louwen F, Klingelhoefer D, Gerber A (November 2013). ... PMID 12533965 . ^ Habek D, Habek JC, Ivanisević M, Djelmis J (2002). "Fetal tobacco syndrome and perinatal outcome". ... PMID 25103301 . ^ Peres GM, Mariana M, Cairrão E (January 2018). "Pre-Eclampsia and Eclampsia: An Update on the Pharmacological Treatment Applied in Portugal" . ... S2CID 25449681 . ^ Salihagić-Kadić A, Medić M, Jugović D, Kos M, Latin V, Kusan Jukić M, Arbeille P (July 2006). ... Retrieved 2020-07-28 . ^ Vásquez-Vivar J, Whitsett J, Derrick M, Ji X, Yu L, Tan S (September 2009).
Cancer In Adolescents And Young Adults Wikipedia

Retrieved 2018-06-08 . ^ a b Fidler, Miranda M; Gupta, Sumit; Soerjomataram, Isabelle; Ferlay, Jacques; Steliarova-Foucher, Eva; Bray, Freddie (2017). ... Archie; Freyer, David R.; Geiger, Ann M. (2016-02-05). "Next steps for adolescent and young adult oncology workshop: An update on progress and recommendations for the future" . ... PMID 26849082 . ^ a b Keegan, Theresa H. M.; Ries, Lynn A. G.; Barr, Ronald D.; Geiger, Ann M.; Dahlke, Deborah Vollmer; Pollock, Bradley H.; Bleyer, W. ... PMID 30480571 . ^ a b c d Warner, Echo L.; Kent, Erin E.; Trevino, Kelly M.; Parsons, Helen M.; Zebrack, Bradley J.; Kirchhoff, Anne C. (2016-04-01). ... G.; Lewis, Denise R.; Harlan, Linda C.; Keegan, Theresa H. M.; Pollock, Bradley H.; Bleyer, W.
Cytokine Release Syndrome Wikipedia

. ^ Shimabukuro-Vornhagen A, Gödel P, Subklewe M, Stemmler HJ, Schlößer HA, Schlaak M, et al. ... PMID 24876563 . ^ a b c Kroschinsky F, Stölzel F, von Bonin S, Beutel G, Kochanek M, Kiehl M, Schellongowski P (April 2017). ... PMID 26189369 . ^ Murphy K, Travers P, Walport M (2007). "Signaling Through Immune System Receptors" . ... Saunders. ISBN 978-0-7216-7996-9 . ^ Wadman M, Couzin-Frankel J, Kaiser J, Matacic C (April 2020). ... August 2014. ^ Fletcher EA, Eltahir M, Lindqvist F, Rieth J, Törnqvist G, Leja-Jarblad J, Mangsbo SM (January 2018).

ARR3, CD19, CXADRP1, SPG7, CXADR, NR1I3, PRKAR1A, CASR, TRIM13, IL6, TWIST1, IFNG, ERBB2, CD226, CARTPT, CST8, MIR146A, MIR34A, KLRK1, MT1E, CCL2, IL17A, TNFRSF9, IL2, IL1B, IL1A, CRP, CD28, KLRC4-KLRK1
Treatment-Resistant Depression Wikipedia

S2CID 2848646 . ^ a b Papakostas, G. I., & Fava, M. (2010). Pharmacotherapy for depression and treatment-resistant depression. ... PMID 16848672 . ^ a b Berman RM, Narasimhan M, Charney DS (1997). "Treatment-refractory depression: definitions and characteristics". ... S2CID 32936223 . ^ Friedman, E. S., & Anderson, I. M. (2011). Managing depression in clinical practice. ... PMID 25341010 . ^ Serafini G, Howland RH, Rovedi F, Girardi P, Amore M (September 2014). "The role of ketamine in treatment-resistant depression: a systematic review" . ... PMID 21184287 . ^ Greden, J., Riba, M., & McInnis, M. (2011). Treatment resistant depression: A roadmap for effective care.

SLC6A4, SLC6A2, VEGFA, MAOA, MAP2K1, RGS4, WDR64, BDNF, SLIT3, OPN3, ZNF385D-AS1, LINC01643, ZNF385D, STAR, TNF, IL6, LRP2, MCF2L, CRP, CREB1, GRIN2B, UGT2B7, TYMS, MLXIP, TXN, CHL1, MYLIP, SMUG1, PYCARD, MARCHF5, ZNF804A, TPH2, MARCHF8, MIRLET7B, MIRLET7C, MIR155, MDD1, ECT, TNFRSF1A, FAS, TRD, TAC1, CACNA1C, CDK9, COMT, CSF2, CYP2C19, CYP2D6, DBT, FKBP5, MSTN, GRM2, HTR1A, IL1B, IL6R, IL18, LAMC2, COX2, NHS, NTRK2, OPRK1, OXTR, POMC, PSMD13, PTGS2, MTCO2P12
Anovulation Wikipedia

Gynecol . 185 (5): 1168–1173. doi : 10.1067/mob.2001.117667 . PMID 11717652 . ^ Filicori M, Flamigni C, Dellai P, et al. (October 1994). ... PMID 14645183 . ^ Freundl G, Bremme M, Frank-Herrmann P, Baur S, Godehardt E, Sottong U (June 1996). ... S.; Braam, S.; Konig, T. E.; Hendriks, M. L.; Hamilton, C. J.; Smeenk, J. M. J.; Koks, C. A. M.; Kaaijk, E. M.; Hompes, P. G. A.; Lambalk, C. B.; van der Veen, F.; Mol, B. W. J.; van Wely, M. (2014). "How long should we continue clomiphene citrate in anovulatory women?"

NR5A2, AMH, FSHR, PRL, MAPK3, NPR2, NPPC, DRD3, ESR2, PHB, KISS1R, DENND1A, BMP15, PAPSS2, VEGFA, TRH, STAU1, BRD2, CYP19A1, DRD2, PCOS1, EPHA7, FURIN, MAP3K8, MPO, MMP9, LOX, LHCGR, INSR, INS, IL1B, AOPEP
Livedo Reticularis Wikipedia

J Am Acad Dermatol . 52 (6): 1009–19. doi : 10.1016/j.jaad.2004.11.051 . PMID 15928620 . ^ Feldaker M, Hines E, Kierland R (1955). "Livedo reticularis with summer ulcerations". ... PMID 13214346 . ^ Gould, Jennifer W.; Helms, Stephen E.; Schulz, Susan M.; Stevens, Seth R. (1998). "Relapsing livedo reticularis in the setting of chronic pancreatitis". ... PMID 843026 . ^ Buckley, Sarah A.; Lessing, Juan N.; Mark, Nicholas M. (2013). "Livedo Reticularis in a Patient with Pheochromocytoma Resolving After Adrenalectomy" . ... Couns . 22 (4): 385–92. PMID 22303799 . ^ Daneshpazhooh M, Nazemi TM, Bigdeloo L, Yoosefi M (2007). ... CS1 maint: DOI inactive as of January 2021 ( link ) ^ a b Tabata, N; Oonami, K; Ishibashi, M; Yamazaki, M (2010). "Livedo Vasculopathy Associated with IgM Anti-phosphatidylserine-prothrombin Complex Antibody" .

CALR, F5, HLA-DPB1, JAK2, PLG, ADA2
- Livedo Racemosa Wikipedia
  
  Livedo racemosa Specialty Dermatology Livedo racemosa is a skin condition with persistent red or violet discoloration, characterised by a broken, branched, discontinuous and irregular pattern. It can be restricted to the limbs or diffuse. It is usually the first sign of a systemic vascular disorder . [1] See also [ edit ] Livedo reticularis Livedoid dermatitis List of cutaneous conditions References [ edit ] ^ "Livedo racemosa: Clinical, laboratory and histopathological findings in 33 patients and literature review" . Journal of the American Academy of Dermatology . 76 (6): AB425. June 2017. doi : 10.1016/j.jaad.2017.06.143 . ISSN 0190-9622 . This dermatology article is a stub . You can help Wikipedia by expanding it . v t e
Hypertensive Heart Disease Wikipedia

Retrieved Nov 11, 2009 . ^ Kearney PM, Whelton M, Reynolds K, Muntner P, Whelton PK, He J (2005). ... PMID 1301257 . ^ Yamasaki N, Kitaoka H, Matsumura Y, Furuno T, Nishinaga M, Doi Y (May 2003). "Heart failure in the elderly" . ... ISBN 978-92-4-156371-0 . ^ Kearney, Patricia M; Whelton, Megan; Reynolds, Kristi; Whelton, Paul K; He, Jiang (2004). ... S2CID 24840738 . } ^ a b c Rabi DM, Khan N, Vallée M, Hladunewich MA, Tobe SW, Pilote L (June 2008). ... PMID 18359324 . ^ Lloyd-Jones D, Adams R, Carnethon M, De Simone G, Ferguson TB, Flegal K, Ford E, Furie K, Go A, Greenlund K, Haase N, Hailpern S, Ho M, Howard V, Kissela B, Kittner S, Lackland D, Lisabeth L, Marelli A, McDermott M, Meigs J, Mozaffarian D, Nichol G, O'Donnell C, Roger V, Rosamond W, Sacco R, Sorlie P, Stafford R, Steinberger J, Thom T, Wasserthiel-Smoller S, Wong N, Wylie-Rosett J, Hong Y (January 2009).

NPPB, ACE, MMP2, AGTR1, AGT, NPR1, CAPN9, FGF21, ITGB1BP2, GDE1, ADRA1A, FENDRR, SOCS3, MIR155, MIR19B1, MIR25, CARMN, PGR-AS1, SLC33A1, SLC8A1, BCL10, TIMP2, REN, PPARA, OGN, MMP9, NR3C2, DPP4, CYP11B2, CTF1, ATM, MHRT
Inclusion Body Myositis Wikipedia

Find sources: "Inclusion body myositis" – news · newspapers · books · scholar · JSTOR ( September 2009 ) ( Learn how and when to remove this template message ) Inclusion body myositis Other names sIBM Specialty Rheumatology Inclusion body myositis ( IBM ) ( / m aɪ oʊ ˈ s aɪ t ɪ s / ) (sometimes called sporadic inclusion body myositis , sIBM ) is the most common inflammatory muscle disease in older adults. [1] The disease is characterized by slowly progressive weakness and wasting of both proximal muscles (closest to the body's midline) and distal muscles (the limbs), most apparent in the finger flexors and knee extensors . [2] IBM is often confused with an entirely different class of diseases, called hereditary inclusion body myopathies (hIBM). [3] [4] The "M" in hIBM is an abbreviation for "myopathy" while the "M" in IBM is an abbreviation for "myositis". ... References [ edit ] ^ Ahmed, Mhoriam; Machado, Pedro M; Miller, Adrian; Spicer, Charlotte; Herbelin, Laura; et, al (March 23, 2016). ... PMID 19080758 . S2CID 16391053 . ^ Kitazawa M, Vasilevko V, Cribbs DH, LaFerla FM (13 May 2009). ... CS1 maint: multiple names: authors list ( link ) ^ Needham M, Mastaglia FL, Garlepp MJ (2007). ... S2CID 41203262 . ^ Broccolini A.; Mirabella M. (2014). "Hereditary inclusion-body myopathies" .

GNE, NT5C1A, APP, TARDBP, HLA-DRB1, SQSTM1, APOE, KHDRBS1, NUP62, DCTN4, GTF2H1, SDC1, CDR3, GSN, MAPT, TRBV20OR9-2, HLA-C, PLAAT4, FYCO1, MSTN, TNFRSF12A, NFAT5, CCR2, UBB, MALAT1, VCP, RBM45, AOC3, DCD, UCN2, DNAJB6, OPTN, KLRG1, MAP1LC3A, LILRB1, KDELR1, ICOSLG, SYNM, ROBO3, DDX58, CHMP1B, PABPC1, TIMP1, RRM2B, TWNK, FOXP3, KRT20, TTR, ACTB, THBS1, CST3, HLA-DQA1, HK1, H1-0, NR3C1, EPHB2, EMD, DES, CD47, TGFB1, CD38, CD36, CD34, MS4A1, CAPN3, BCL2, AOC2, HLA-DRB3, HMGB1, IFN1@, IFNG, TRIM21, AGER, MOK, PTPRC, PSME1, PSMB10, MAPK1, POLG, PMP22, MMP9, MMP1, MLF1, LMNA, IL6, IL1B, LOC102723996
- Inclusion Body Myositis Omim
  
  Description Sporadic inclusion body myositis (IBM) is the most common age-related muscle disease in the elderly that results in severe disability. Although traditionally considered an inflammatory myopathy, it is now considered to be more consistent with a myodegenerative disease (Sugarman et al., 2002; Askanas and Engel, 2006). Clinical Features The vast majority of IBM occurs sporadically; however, rare familial occurrence has been reported. Inclusion body myositis is a slowly progressive inflammatory myopathy characterized clinically by weakness of the proximal parts of the limbs, diminished deep tendon reflexes, dysphagia, and mixed myopathic and neurogenic changes on electromyography. Baumbach et al. (1990) reported the first familial cases. Six persons in 2 generations were affected in an autosomal dominant pattern of inheritance.
- Inclusion Body Myositis Orphanet
  
  Inclusion body myositis (IBM) is a slowly progressive degenerative inflammatory disorder of skeletal muscles characterized by late onset weakness of specific muscles and distinctive histopathological features. Epidemiology IBM has a highly variable prevalence according to geographic, ethnic and age criteria. Prevalence in the general population ranges from 1:1,000,000 to 1:14,000 but a three-fold increase is observed when considering only a population over 50 years. Underdiagnosis may be an explanation for the high ethno-geographic variation. Male-to-female ratio is 2:1 on average (0.5 to 6.5:1). Clinical description IBM onset is over 50 years but may also occur earlier, in the 5th decade.
- Idiopathic Inflammatory Myopathy Medlineplus
  
  Idiopathic inflammatory myopathy is a group of disorders characterized by inflammation of the muscles used for movement (skeletal muscles). Idiopathic inflammatory myopathy usually appears in adults between ages 40 and 60 or in children between ages 5 and 15, though it can occur at any age. The primary symptom of idiopathic inflammatory myopathy is muscle weakness, which develops gradually over a period of weeks to months or even years. Other symptoms include joint pain and general tiredness (fatigue). There are several forms of idiopathic inflammatory myopathy, including polymyositis, dermatomyositis, and sporadic inclusion body myositis. Polymyositis and dermatomyositis involve weakness of the muscles closest to the center of the body (proximal muscles), such as the muscles of the hips and thighs, upper arms, and neck.
- Inclusion Body Myositis Gard
  
  Inclusion body myositis (IBM) is a progressive muscle disorder characterized by muscle inflammation, weakness, and atrophy (wasting). It is a type of inflammatory myopathy . IBM develops in adulthood, usually after age 50. The symptoms and rate of progression vary from person to person. The most common symptoms include progressive weakness of the legs, arms, fingers, and wrists. Some people also have weakness of the facial muscles (especially muscles controlling eye closure), or difficulty swallowing (dysphagia). Muscle cramping and pain are uncommon, but have been reported in some people.
Hot Flash Wikipedia

. ^ Spetz AC, Zetterlund EL, Varenhorst E, Hammar M (November–December 2003). "Incidence and management of hot flashes in prostate cancer". ... PMC 6287288 . PMID 20566620 . ^ L'Hermite M (August 2013). "HRT optimization, using transdermal estradiol plus micronized progesterone, a safer HRT" . ... S2CID 37254359 . ^ Bergman Jungeström M, Thompson LU, Dabrosin C (February 2007). ... PMID 17289903 . ^ a b Touillaud MS, Thiébaut AC, Fournier A, Niravong M, Boutron-Ruault MC, Clavel-Chapelon F (March 2007). ... Journal of Medicinal Food . 6 (1): 1–11. doi : 10.1089/109662003765184697 . PMID 12804015 . ^ Lock M (May–August 1994). "Menopause in cultural context".

ESR2, CYP2D6, ESR1, TAC3, TACR3, CYP19A1, CYP1B1, SHBG, CYP3A4, CALCA, ADIPOQ, SLC6A2, CCHCR1, PPIG, SELENBP1, TNF, SLC50A1, SULT1A1, SLC6A4, AHR, LEP, S100A12, NOS3, ANXA13, LALBA, CXCL8, HIF1A, GAD1, CYP17A1, CYP1A1, COMT, UCN3
- Hot Flashes Mayo_clinic
  
  Overview A hot flash is the sudden feeling of warmth in the upper body, which is usually most intense over the face, neck and chest. Your skin might redden, as if you're blushing. A hot flash can also cause sweating. If you lose too much body heat, you might feel chilled afterward. Night sweats are hot flashes that happen at night, and they may disrupt your sleep. Although other medical conditions can cause them, hot flashes most commonly are due to menopause — the time when menstrual periods become irregular and eventually stop. In fact, hot flashes are the most common symptom of the menopausal transition.
Cervical Pregnancy Wikipedia

PMID 27408132 . ^ Kraemer B, Abele H, Hahn M, Wallwiener D, Rajab TK, Hornung R (2008). ... PMID 18710714 . ^ Corticelli A, Grimaldi M, Caporale E (2008). "Conservative management of cervical ectopic pregnancy: case report and review of literature". ... PMID 19205450 . ^ Nakao Y, Yokoyama M, Iwasaka T (2008). "Uterine artery embolization followed by dilation and curettage for cervical pregnancy". ... PMID 17630167 . ^ Kotzbach R, Szymański M, Korenkiewcz J, Wasilewski Z, Szymański W (2005).

MTX1
Granulomatous Meningoencephalitis Wikipedia

J Am Vet Med Assoc . 225 (8): 1211–6, 1196. doi : 10.2460/javma.2004.225.1211 . PMID 15521442 . ^ Suzuki M, Uchida K, Morozumi M, Yanai T, Nakayama H, Yamaguchi R, Tateyama S (2003). ... R.; Pedersen, N. C.; Ruhe, A.; Wallace, M.; Neff, M. W. (April 2010). "Necrotizing meningoencephalitis of Pug Dogs associates with dog leukocyte antigen class II and resembles acute variant forms of multiple sclerosis".
Triangle Disease Wikipedia

.; Cant, A.; Matthews, H.; Santibanez Koref, M.; Simon, A.; Korolchuk, V.; Lenardo, M.; Hambleton, S.; Su, H. (2014). ... H.; Fuchs, S.; Zenke, S.; Firat, E.; Pessach, V. M.; Borkhardt, A.; Rakhmanov, M.; Keller, B.; Warnatz, K.; Eibel, H.; Niedermann, G.; Elpeleg, O.; Ehl, S. (20 November 2014).
Holmes Tremor Wikipedia

The treatment of choice is complete removal of the tumor. [2] Removing the tumor can result in elimination or better control of the tremors. [2] Other treatment options involve coping strategies such as avoiding movements or actions that worsen tremors. [1] Patients suffering from Holmes tremors can also benefit from using larger utensil handles and wrist weights. [1] There are also some pharmacological treatments, but they are not very effective. [1] References [ edit ] ^ a b c d e f g h i j k l m n o p q r s Puschmann, A; Wszolek, ZK (2011). ... PMID 24926407 . ^ a b c d Buijink, A; Contarino, M; Koelman, J; Speelman, J; van Rootselaar, A (2012). ... Movement Disorders . 26 (10): 1957–1959. doi : 10.1002/mds.23704 . PMID 21542020 . Kim, M C; B C Son; Y Miyagi; J-K Kang (2002). ... PMC 1738060 . PMID 12235320 . Sheppard, Gordon M G; Erik Tauboll; Soren Jacob Bakke; Rolf Nyberg-Hansen (1997).

ABCB7, GABRA4, UROC1, CACNA2D2, SOX2, SLC2A1, SCN8A, ARCN1, NPC1, GABRA1, EGR3, CP, CACNB4, CACNA1A, ATP7A, HCN1, CIT, GK5
Dental Subluxation Wikipedia

. ^ Lecky F, Russell W, Fuller G, McClelland G, Pennington E, Goodacre S, Han K, Curran A, Holliman D, Freeman J, Chapman N, Stevenson M, Byers S, Mason S, Potter H, Coats T, Mackway-Jones K, Peters M, Shewan J, Strong M (January 2016). ... Retrieved 2018-11-12 . ^ Azami-Aghdash S, Ebadifard Azar F, Pournaghi Azar F, Rezapour A, Moradi-Joo M, Moosavi A, Ghertasi Oskouei S (2015).
Endogenous Depression Wikipedia

"Endogenous Versus Exogenous: Still Not the Issue" . Medscape . ^ Nyhuis PW, Specka M, Gastpar M (2006). "Does the antidepressive response to opiate treatment describe a subtype of depression?". ... PMID 7714228 . ^ Nyhuis, P.W.; Specka, M.; Gastpar, M. (2006). "P.2.b.014 Does the antidepressive response to opiate treatment describe a subtype of depression?".

TPH2, DISC1, CRH, POMC, ADORA2A, PTGS2, MIF, MTHFR, NOS1, NPY, NPY1R, PDCD2, PON1, RELN, SELP, S100A10, ALB, SLC6A4, SNAP25, SOD1, TG, PDCD6, CADM1, GHRL, DIXDC1, KCNK2, IL6, IFNA2, FTH1, APP, BDNF, CNR2, CREBBP, CSF3, DRD5, ACSL4, HTR7, FGFR1, FGF2, FTL, GLO1, GNAS, GNB1, NR3C1, GSK3A, GSK3B, HAP1, MORC1, DBH, COMT, HLA-B, HLA-C, ECT
Orthostatic Hypertension Wikipedia

H.; Sun, K.; Zhou, X. L.; Zhang, H. M.; Wu, H. Y.; Hui, R. T. (2011). "Association of orthostatic hypertension and hypotension with target organ damage in middle and old-aged hypertensive patients". ... L.; Zevin, S.; Carlsen, S.; Wright, J.; Schambelan, M.; Cheitlin, M. (1996). "Orthostatic hypertension due to vascular adrenergic hypersensitivity". ... R.; Alonso, A.; Gottesman, R. F. ; Rose, K. M.; Aric Study, I. (2011). "Postural changes in blood pressure and incidence of ischemic stroke subtypes: The ARIC study" .

ADRB1, ACE, TRIP12
Superficial Thrombophlebitis Wikipedia

. ^ a b Decousus H, Quéré I, Presles E, Becker F, Barrellier MT, Chanut M, Gillet JL, Guenneguez H, Leandri C, Mismetti P, Pichot O, Leizorovicz A (February 2010). ... Annals of Internal Medicine . 152 (4): 218–24. doi : 10.7326/0003-4819-152-4-201002160-00006 . PMID 20157136 . ^ Creager M, Loscalzo J, Beckman JA (30 August 2012). ... PMID 2133604 . ^ a b Decousus H, Epinat M, Guillot K, Quenet S, Boissier C, Tardy B (September 2003). ... Chest . 133 (6 Suppl): 454S–545S. doi : 10.1378/chest.08-0658 . PMID 18574272 . ^ Di Nisio M, Wichers IM, Middeldorp S (February 2018).
Tropical Eosinophilia Wikipedia

Retrieved 2011-04-16 . ^ a b c d Yong, M.; Marshall, C.; Eisen, D. (2007). ... Annual Review of Medicine . 43 : 417–424. doi : 10.1146/annurev.me.43.020192.002221 . PMID 1580599 . ^ Muck, A.; Pires, M.; Lammie, P. (2003). "Influence of infection with non-filarial helminths on the specificity of serological assays for antifilarial immunoglobulin G4" (PDF) . ... PMID 16582677 . ^ McLaughlin, S.; Radday, J.; Michel, M.; Addiss, D.; Beach, M.; Lammie, P.; Lammie, J.; Rheingans, R.; Lafontant, J. (2003).

IL5, GATA3, RAG2, SHARPIN, TLR4, TLR2, MYD88, PTGDS, SERPINA1, SIGLEC8, GGTLC1, IL33, JAK2, IL25, MYDGF, HPGDS, POSTN, PTX3, FOXN1, P2RY6, STAT3, RNASE3, RNASE2, LOC102724197
Roussy–lévy Syndrome Wikipedia

PMID 12428130 . ^ a b Auer-Grumbach, M.; Strasser-Fuchs, S.; Wagner, K.; Körner, E.; Fazekas, F. (1998). ... G.; Chauvin, F.; Ollagnon-Roman, E.; Bost, M.; Latour, P.; Bonnebouche, C.; Gonnaud, P. M.; Bady, B.; Chazot, G.; Vandenberghe, A. (1996). ... P.; Cossette, L.; Brunet, D.; Vanasse, M.; Lemieux, B.; Mathon, G.; Puymirat, J. (1999).

MPZ, PMP22, LRSAM1, FIG4, SLC25A46, HOXD10, TRPV4, MORC2, COX6A1, NDRG1, SBF2, SH3TC2, DHTKD1, GDAP1, KIF1B, MFN2, SLC12A6, DYNC1H1, MTMR2, NEFL, LMNA, GJB1, GARS1, EGR2, FGD4, FXN
- Roussy Levy Syndrome Gard
  
  Roussy Levy syndrome is a term used to describe a neuromuscular disorder that typically becomes apparent during early childhood. This syndrome is considered a form of Charcot-Marie-Tooth (CMT) disease . Individuals with this disorder have clinical symptoms similar to Charcot-Marie-Tooth (CMT) disease type 1 , which is characterized by muscle weakness and atrophy, poor judgement of movement (sensory ataxia), absent reflexes (areflexia) of the lower legs and hands, and abnormally high arches of the feet (pes cavus or "clawfoot"). Additional features of rhythmic shaking (static tremor) in the hands and an unsteady gait ( gait ataxia ) are specific to Roussy Levy syndrome. This disorder is caused by issues with nerve conduction and sensory dysfunction.
- Roussy-Levy Hereditary Areflexic Dystasia Omim
  
  INHERITANCE - Autosomal dominant SKELETAL Spine - Kyphoscoliosis may be present Feet - Pes cavus - Hammer toes - Foot deformities NEUROLOGIC Peripheral Nervous System - Delayed motor development - Distal limb muscle weakness due to peripheral neuropathy - Distal limb muscle atrophy due to peripheral neuropathy - Ataxic gait - Distal sensory impairment - Areflexia - Hyporeflexia - Upper limb postural tremor - Upper limb action tremor - Decreased motor nerve conduction velocity (NCV) (less than 20 m/s) - Segmental demyelination/remyelination on nerve biopsy - Decreased number of myelinated fibers - Hypertrophic nerve changes - 'Onion bulb' formations may be present IMMUNOLOGY - Foot ulcerations - Foot infections leading to amputation MISCELLANEOUS - Onset in early childhood - Usually begins in feet and legs (peroneal distribution) - Upper limb involvement usually occurs later - Slowly progressive - Allelic disorders with overlapping phenotypes include Charcot-Marie-Tooth disease type 1 (CMT1B, 118200 and CMT1A, 118220 ) and Dejerine-Sottas syndrome (DSS, 145900 ) MOLECULAR BASIS - Caused by mutation in the myelin protein zero gene (MPZ, 159440.0021 ) - Caused by mutation in the peripheral myelin protein-22 gene (PMP22, 601097.0001 ) ▲ Close
- Roussy-Lévy Syndrome Orphanet
  
  A rare demyelinating hereditary motor and sensory neuropathy characterized by prominent gait ataxia, pes cavus, tendon areflexia, distal limb weakness, tremor in the upper limbs, distal sensory loss, kyphoscoliosis, and progressive muscle atrophy. The disease becomes symptomatic in infancy or childhood, mode of inheritance is autosomal dominant.
Diabetic Foot Wikipedia

It is unclear if local antibiotics improve outcomes after surgery. [12] References [ edit ] ^ Hefni, Abd Al-Hamead; Ibrahim, Al-Metwally R; Attia, Khaled M.; Moawad, Mahmoud M.; El-ramah, Ayman F.; Shahin, Mohamed M.; Al-Molla, Mahmoud; Abd Al-Satar, Lotfi. ... Retrieved 23 November 2015 . ^ Darlington, C., Kumar, S., Jagdish, S., Sridhar, M. Evaluation of Serum Vitamin D Levels in Diabetic Foot Infections: A Cross-Sectional Study in a Tertiary Care Center in South India.

MAPK14, UNC5D, TRDN, CRP, TGFB1, BTK, SIRT1, FGF23, SELENBP1, KL, ADIPOQ, CCHCR1, TLR9, TGFBR2, SMIM10L2A, MIR146A, MIR203A, MIR375, SMIM10L2B, C20orf181, TLR2, TGFB3, TGFBR1, LPA, DEFB4A, DPP4, EDN1, HSPA1B, CXCL8, INS, NOS3, TGFB2, TNFRSF11B, SERPINE1, PLG, PON1, CXCL5, SHBG, RN7SL263P