-
Abortion In Georgia (U.s. State)
Wikipedia
Jones ruled in favor of the injunction to block enforcement in his decision in October 2019, stating "By banning pre-viability abortions, H.B. 481 violates the constitutional right to privacy, which, in turn, inflicts per se irreparable harm on Plaintiffs." [28] Statistics [ edit ] In the period between 1972 and 1974, the state had an illegal abortion mortality rate per million women aged 15 – 44 of between 0.1 and 0.9. [29] In 1990, 796,000 women in the state faced the risk of an unintended pregnancy. [30] In 2010, the state had eight publicly funded abortions, of which all eight were federally funded. [31] In 2014, 48% of adults said in a poll by the Pew Research Center that abortion should be legal in all or most cases. [1] According to a 2020 study, the 22-week law reduced the number of abortions after 21 weeks. [32] Number of reported abortions, abortion rate and percentage change in rate by geographic region and state in 1992, 1995 and 1996 [33] Census division and state Number Rate % change 1992–1996 1992 1995 1996 1992 1995 1996 South Atlantic 269,200 261,990 263,600 25.9 24.6 24.7 –5 Delaware 5,730 5,790 4,090 35.2 34.4 24.1 –32 District of Columbia 21,320 21,090 20,790 138.4 151.7 154.5 12 Florida 84,680 87,500 94,050 30 30 32 7 Georgia 39,680 36,940 37,320 24 21.2 21.1 –12 Maryland 31,260 30,520 31,310 26.4 25.6 26.3 0 North Carolina 36,180 34,600 33,550 22.4 21 20.2 –10 South Carolina 12,190 11,020 9,940 14.2 12.9 11.6 –19 Virginia 35,020 31,480 29,940 22.7 20 18.9 –16 West Virginia 3,140 3,050 2,610 7.7 7.6 6.6 –14 Number, rate, and ratio of reported abortions, by reporting area of residence and occurrence and by percentage of abortions obtained by out-of-state residents, US CDC estimates Location Residence Occurrence % obtained by out-of-state residents Year Ref No.
- Binswanger's Disease Wikipedia
-
Inflammatory Breast Cancer
Wikipedia
Breast Cancer Research and Treatment . 95 (3): 219–28. doi : 10.1007/s10549-005-9002-1 .ALK, RHOC, EGFR, ESR1, ERBB2, TP53, PGR, CCN6, EGF, PIK3CA, XIAP, CD274, ERBB3, CAV1, MTCO2P12, NFKB1, PTGS2, COX2, CTNNB1, EZH2, NOTCH3, TNFSF10, PIK3CG, MYC, EIF4G1, STAT3, PIK3CB, PIK3CD, IL13, RIPK2, CD68, AHR, AKT1, APC, SDC1, MUC1, CXCL8, PTK2, PTEN, COL1A1, GLI1, VEGFA, IL10, FOXP3, INPP4B, ARHGEF7, GPRC5A, SLIT2, ADM, TP63, LGR5, TFF1, TGFA, THBD, TNF, TP73, SUMO1, UVRAG, VDR, VIM, WNT1, WNT5A, CXCL14, CXCR4, H3-4, IFITM1, SLC9A3R1, B3GAT1, APOBEC3B, TAS2R64P, MIR126, MIR150, MIR155, MIR15A, MIR181C, MIR19A, MIR205, MIR21, MIR26B, MIR30B, MIR335, MIR337, MIR451A, HOTAIR, MIR1303, MIR210HG, TMX2-CTNND1, MIR10B, ARHGAP24, PUM1, FTO, KIAA0100, HDAC6, SCGB1D2, NES, CKAP4, KCNQ1OT1, UBE2C, TJP3, AGO2, REM1, ASCC1, PRLH, NUSAP1, MED15, WWOX, KRT20, DGCR8, STAT6, PMAIP1, ST14, IDH2, CST6, CTNND1, CTSB, CTSV, CYP1B1, DHCR24, DLX4, ECT2, ERBB4, F2R, FGFR1, GLO1, GPX3, HAGH, HGF, CSF1R, CSF1, CPB2, BRCA2, AGT, ALDH1A1, AREG, BAX, BCL2, BRCA1, CD1A, COL10A1, MS4A1, CD40, CD44, CDK2, CCR7, COL5A1, HHEX, IL6, SIAH2, IL13RA1, PAK1, PDGFRA, PDGFRB, PARP1, CTSA, PPP3CA, PRKCZ, MAP2K7, PSEN2, RARRES1, RBM3, RHO, RPE65, S100A7, SELE, TNFRSF11B, NOTCH1, NFKB2, MARCKS, IL13RA2, IL18, KDR, KRT18, KRT81, LMNA, MCL1, MYB, MMP1, MMP2, MMP3, MMP11, MMP12, MUC4, H3P40
-
Achilles Tendon Rupture
Wikipedia
. ^ Khan-Farooqi W, Anderson RB (April 28, 2010). "Achilles tendon evaluation and repair" .
-
Pulmonary Fibrosis
Wikipedia
. ^ "www.spirXpert.com" . Archived from the original on 28 January 2010. ^ King TE Jr; Bradford WZ; Castro-Bernardini S; et al.TGFB1, CCN2, EDN1, IL1B, MMP9, NFE2L2, IL4, CCL2, IL13, TIMP1, AGT, CSF2, HPS1, FAM13A, DSP, AP3B1, ABCA3, FN1, HGF, TNF, IL6, HMGB1, TGFA, STAT3, IGF1, FGF2, CXCL8, MTOR, MMP2, SOD1, CCL11, CCR2, SERPINA1, SPP1, AREG, MIR326, ADIPOQ, HMOX1, STAT6, MIR30A, MECP2, EGF, COL3A1, MIR29C, CAT, SOD3, BMP7, FGF1, ELN, CEBPB, FGF7, CCL5, GREM1, CCL4, CCR3, PARP1, SMAD7, ACTA2, LAMB1, PDGFA, IL1RN, IL5, CSF3, MIR140, MIR125A, CYSLTR2, CEBPA, MIR26B, CMA1, FYN, MIR200C, IL12B, MIR122, MIR345, CALCA, SKIL, MIR10A, PDGFB, PTX3, SERPINE1, MIR101-1, CXCL2, MIR425, MIR378A, CCL3, CFD, EGFR, IL17A, PTGS2, AGER, COL1A1, ACE, PLAU, SMAD4, MMP1, SCGB1A1, IL1A, ELANE, CD36, TGFBR2, COL1A2, PDGFRA, CCL17, CCL22, ANXA1, HSP90AB1, CDH1, HABP2, STAT1, ICAM1, MFN2, CYSLTR1, TERT, B3GAT1, HSPD1, MUC5B, CCR4, BAD, VWF, NKX2-5, CCR8, SFTPC, MUC5AC, CAV1, HPS4, RTEL1, TERC, PARN, IRF5, SFTPA2, SFTPA1, FAM111B, HLA-DRB1, TINF2, DKC1, HLA-DPB1, CTLA4, CFTR, DCTN4, PRTN3, PRKCD, FASLG, FAS, ACTB, BTNL2, HIF1A, IL10, MRPS22, PIK3CD, PIK3CG, PIK3CB, PIK3CA, RNF168, SMAD2, NLRP3, STN1, SP110, BMP15, NHP2, HLA-DPA1, KIAA0319L, DPP9, RCBTB1, SMAD3, NOP10, NR5A1, NUP107, FSHR, THOC2, NOX4, PLEC, PSMC3IP, RASGRP1, ALMS1, ASAH1, POSTN, NKX2-1, CASP10, CLCA4, STX1A, CCR6, ATP11A, SMN2, SMN1, SPIDR, CTNNB1, PTPN22, MIR21, F2R, VEGFA, TP53, LOX, AKT1, GABPA, CXCR4, TLR4, FOSL2, PLG, COPD, ACE2, THY1, ANGPT1, TNC, SIRT3, MMRN1, GLI1, DECR1, BLM, LPAR1, CXCL12, VIM, CEL, FSTL1, LOXL2, SLC27A5, PARP9, PRKAB1, IL33, JAK2, PRKAA1, PRKAA2, TM7SF2, IFNG, MTCO2P12, SIRT1, SFTPD, COX2, LGALS3, MMP12, CCL18, MMP7, REN, S100A4, HSPA4, NR1I2, PTEN, MUC1, MIRLET7D, ZEB1, MIR34A, OGG1, ADAM17, MSC, VTN, CCN4, YY1, PPARG, SPHK1, SEMA7A, NT5E, DNM1L, SMUG1, TOP1, BRD4, SLC52A2, HSPB3, PINK1, TMED7, SPARC, PDGFRB, TGFB3, GDF15, PHEX, MAPK3, KLF4, HSP90AA1, DNER, MAPK14, MARK2, TMED7-TICAM2, F2RL1, TICAM2, BLMH, TSLP, GLB1, PWAR1, GSK3B, FOXO3, EGR1, CD44, DDIT3, DCN, DNMT1, ING4, MAPK1, ELK1, MAPK7, MAPK8, PSMD9, TLR2, LILRB1, PTK2, PTPN11, DCTN6, IL7, TSC1, CXCR6, FOLR2, PDCD1, LCLAT1, ZNRD2, FOXM1, WNT1, SLC52A1, PF4, ATF3, SSRP1, AHSA1, BDNF, STING1, UTRN, SIRT6, TXN, FASN, TWIST1, PTPRC, MIR145, HPGDS, MIR154, CD274, CST3, CHI3L1, NLRP1, CSF1R, CSF1, SLC2A1, CRYAB, CRMP1, SNAI1, CRK, SOAT1, MIR29A, SOD2, CREBBP, MIR29B1, MIR29B2, STAT4, CNR2, LTB4R, DAPK2, SDC2, SDC1, TGFBI, MOK, THBD, PRKN, METAP2, IL37, MIR155, MIR15A, MIR18A, DPP4, CX3CL1, SERPINH1, RIPK3, POLDIP2, RNF19A, TFF1, MIR200B, NUDT21, CDH11, FOS, BACH1, SOCS1, ANXA2, LGALS1, SQSTM1, JAG1, LOXL1, PERCC1, TNFSF14, HDAC4, MARCO, CTHRC1, BECN1, DENR, MIP, RBM45, HAS2, CXCL16, ANPEP, MMP13, ROCK2, LDHA, HSPB1, SETD7, S1PR2, IL9, MAPKAPK2, H3P23, CCN1, MAP1LC3B, IL18, IRAK1, HSPB2, KL, IGFBP3, GRAP2, MCU, IFI27, ITGB6, ADORA2B, LTBP4, ST2, MYD88, MPO, FST, GLI2, GPX4, P2RX7, ANXA5, TSPAN1, YAP1, CBLIF, AIMP2, GLRX, TFEB, GAS6, IL18BP, OSM, NTN1, MIR323A, RAPGEF3, MIR542, MIR1224, CHP1, TMPRSS11D, ABCG2, AIM2, GGPS1, DUSP10, TRIM16, MIR328, MIR96, LINC02605, KLF2, MIR34B, P2RX2, MIR31, MERTK, FBLN5, KAT5, CXCL13, ATG7, SLIT2, H3P11, TRIP10, MIR30D, P2RX5-TAX1BP3, ATG5, MIR541, RAMP2, ATP6AP2, MIR506, PPARGC1A, TNFSF13B, GAB2, MIR5100, MIR486-1, MIR454, MIR455, DNM3OS, CAPN9, FAM72A, NEU3, FAM72B, MIR627, HDAC6, SPATA2, RPL17-C18orf32, JTB, KHDRBS1, PLF, MIR448, PLXNC1, MIR4516, MIR449A, SIRT1-AS, CDR1-AS, PCLAF, ABCG1, H4C15, RGS6, LRPPRC, MIR503, CD226, TMX2-CTNND1, B3GAT3, ATG4B, ACKR3, TRPV4, GER, H4-16, SLCO6A1, ROMO1, TRIB3, PRX, MRTFA, RCN3, RMDN2, ASPRV1, KMT5AP1, PCBP4, TCIM, SULF1, SPHK2, RHOV, TMPRSS4, PARD3, PGAM5, MYDGF, SULF2, HDAC8, PAG1, IL26, MFN1, PRSS55, ZEB1-AS1, RXFP1, FKBP10, SCGB3A2, SMOC2, SPZ1, RETNLB, ING5, CRISPLD2, LOXL3, HAVCR2, CD276, ADAM33, WNT10A, ZC3H12A, GRHL2, NAF1, MMP28, ELOVL6, TRPM8, WNK1, CDCP1, IL25, MUC16, PRRT2, DEPTOR, NREP, SEMA4A, IFIH1, IL17F, UCN3, FBXO32, SYBU, EBF3, ELMOD2, ADGRE2, TBK1, SETD2, IL17B, BBC3, LATS2, FGF21, KISS1R, PHGDH, MIR185, PART1, MIR193A, MIR19A, MIR19B1, MIR205, MIR210, PRDX5, NUP62, MIR221, DDAH1, PIGN, MIR27A, TRAM1, MIR27B, CBX5, HEY1, PPP1R13B, MIR301A, MIR146A, DELEC1, RMDN3, MIR144, SARS2, EGLN1, ROBO4, NPNT, IFNL3, POLE3, LAMA1, PWAR4, GSTK1, SIRT7, GTSE1, MALAT1, FENDRR, SNX9, CSAD, CHST15, CRNKL1, TNFRSF12A, MIR126, MIR130A, MIR130B, MIR139, RMDN1, ADIPOR1, MIR141, MIR142, FOXP3, GORASP1, SFRP4, CRLF1, GRP, FUT8, GAB1, GATA3, GLS, CCR10, CXCR3, GPT, GTF2H1, HPGD, HDAC2, CFHR1, NRG1, HLA-A, HLA-B, HLA-C, HLF, FOSB, FLT3LG, FLT3, FLT1, ESR1, ETS2, F2, F2RL2, F3, F10, FAP, FCGR3A, FCGR3B, FGF13, FGFR2, FGL1, VEGFD, FOXF1, FOXD1, NR4A1, HSPA1A, ERN1, JUNB, ITGAE, ITGAX, ITGB3, EIF6, IVD, JAK1, JUN, JUND, HSPA1B, KCNMB1, LEP, LMNA, LRP6, LTBR, LUM, MARCKS, ITGA9, ISG20, ITGA6, INPP5D, HTR2A, ID1, IFNB1, IGF2, IGFBP2, IGFBP5, IL2, IL2RA, IL4R, IL6ST, IL7R, IL11, IL11RA, IL15, TNFRSF9, FBL, ERBB3, MEN1, CAST, KLF9, TSPO, C3, C3AR1, C5AR1, VPS51, DDR1, CASP1, CDKN2A, RUNX2, RUNX3, CBS, CD28, CD59, ADGRE5, CD151, BSG, BMPR2, BMP3, BGN, ADA, ADAM10, ADM, AEBP1, AGTR1, AIF1, AKT2, ALB, ANGPT2, ANXA11, APOA1, APRT, ARG1, BARD1, BCL2, CDH5, CDKN2B, ERBB2, E2F4, CYP27B1, DES, NQO1, DIO2, DNAH8, ARID3A, E2F1, ECE1, CHAT, ECT2, S1PR3, EFNB2, EPHA2, EIF4E, EP300, STX2, CYP2E1, CYLD, CX3CR1, CTSK, CHIT1, CHRM3, CHRNA4, CLU, ACKR2, CNR1, COL11A2, COX8A, CREB1, ATF2, CRIP1, CRYZ, CSNK1D, CSRP1, CTNND1, MCAM, MFAP1, XPR1, TNFRSF1A, TGFB2, TGFBR1, THAS, THBS1, TIAM1, TIMP3, TLR3, TNNC1, WT1, TPH1, CRISP2, TRPC6, TSC2, TNFSF4, VCAM1, VDR, TMBIM6, PPP1R11, TRBV20OR9-2, TAZ, SHH, SIX1, SKI, SKP2, SNAI2, SNCA, SOX2, SPINT1, SRC, SREBF1, SRF, STC1, STK11, SYT1, TAGLN, TRPV1, XIST, SFRP1, MBD2, AOC3, EIF3A, MBTPS1, TNFSF10, CCN6, CCN5, SGPL1, TRPA1, FOSL1, F2RL3, TM4SF5, SYT7, P2RX6, ATG12, LPAR2, IL1RL1, BHLHE40, ENC1, RASAL1, ULK1, HMGA2, H3-4, H4C9, FZD7, H4C1, H4C4, H4C6, H4C12, H4C11, H4C3, H4C8, H4C2, H4C5, H4C13, H4C14, ACO2, SELPLG, MID1, PAWR, P2RX3, P2RX4, P2RX5, P2RY1, P2RY2, P4HB, FURIN, PCNA, PIP, PDE1A, PDK1, PECAM1, ATP8B1, ABCB1, PHB, SERPINE2, P2RX1, DDR2, NTF3, NOTCH4, MAP3K11, MMP3, MMP10, MMP14, MMP19, MPG, MRC1, MSR1, MST1, NEDD8, NEU1, NFKB1, NINJ1, NM, NOTCH1, PIN1, PLA2G4A, SELE, ROCK1, RASA1, RBP2, OPN1LW, RELA, RGS2, BRD2, RNH1, ROS1, PLAUR, RPL17, S100A6, S100A9, SARS1, SERPINB3, CCL24, SDC4, RARRES2, RAG2, RAF1, RAC2, PLK1, POU2AF1, PPL, PRH1, PRH2, PRKCA, PRKCB, PRKDC, EIF2AK2, PTCH1, PTGER4, PTHLH, PTPN13, PTPRJ, RAC1, H3P10
- Anemia In Pregnancy Wikipedia
-
Central Pontine Myelinolysis
Wikipedia
Imaging by MRI typically demonstrates areas of hyperintensity on T2-weighted images. [ citation needed ] Treatment [ edit ] To minimise the risk of this condition developing from its most common cause, overly rapid reversal of hyponatremia, the hyponatremia should be corrected at a rate not exceeding 10 mmol/L/24 h or 0.5 mEq/L/h; or 18 mEq/L/48hrs; thus avoiding demyelination. [27] No large clinical trials have been performed to examine the efficacy of therapeutic re-lowering of serum sodium, or other interventions sometimes advocated such as steroids or plasma exchange. [27] Alcoholic patients should receive vitamin supplementation and a formal evaluation of their nutritional status. [28] [29] Once osmotic demyelination has begun, there is no cure or specific treatment.
-
Salicylate Poisoning
Wikipedia
.; Rutka, John A. (2004). Ototoxicity . PMPH-USA. p. 28. ISBN 9781550092639 . Archived from the original on 10 September 2017 .
-
Hiatal Hernia
Wikipedia
PubMed Health . Archived from the original on 28 April 2017 . Retrieved 6 May 2017 . ^ Chang SW, Lee HC, Yeung CY, Chan WT, Hsu CH, Kao HA, Hung HY, Chang JH, Sheu JC, Wang NL (2010).ATP7A, COL1A1, GPHN, WDR4, AFG3L2, SPECC1L, NIPBL, TPRKB, OSGEP, NUP133, NUP107, PORCN, SLC2A10, TUBB6, ATAD1, WDR73, TP53RK, ADAMTS2, SLC6A5, LAGE3, PTCH1, COL5A1, COL5A2, TNXB, GLRB, GLRA1, ALDH18A1, SMARCB1, TCF4, TGFB3, CHST14, IL12B, HLA-DMA, GER, FUT3, ECI1, ADGRV1, CYP2C19, COL3A1
-
Reactive Hypoglycemia
Wikipedia
It might be an " adrenergic postprandial syndrome" — blood glucose levels are normal, but the symptoms are caused through autonomic adrenergic counterregulation. [28] Often, this syndrome is associated with emotional distress and anxious behaviour of the patient. [13] This is often seen in dysautonomic disorders as well.
-
Agrammatism
Wikipedia
"Linguistic complexity and frequency in agrammatic speech production" (PDF) . Brain and Language . 109 : 18–28. doi : 10.1016/j.bandl.2008.12.004 .
-
Brainstem Death
Wikipedia
No testing of testable brain stem functions such as oesophageal and cardiovascular regulation is specified in the UK Code of Practice for the diagnosis of death on neurological grounds. There is published evidence [28] [29] [30] strongly suggestive of the persistence of brainstem blood pressure control in organ donors .
-
Reduced Affect Display
Wikipedia
They seem to lack feelings altogether, although this may actually be because of their inability to express emotion rather than from an absence of emotion altogether". [28] Alexithymic patients however can provide clues via assessment presentation which may be indicative of emotional arousal . [29] "If the amygdala is severed from the rest of the brain, the result is a striking inability to gauge the emotional significance of events; this condition is sometimes called 'affective blindness'". [30] In some cases, blunted affect can fade, but there is no conclusive evidence of why this can occur.
-
Cataplexy
Wikipedia
"Emerging Therapies in Narcolepsy-Cataplexy" . Sleep . 28 (6): 754–763. doi : 10.1093/sleep/28.6.754 .
-
Blepharitis
Wikipedia
In this study, researchers provide a diagnosis of the disease and propose diagnostic criteria of Demodex blepharitis. [28] References [ edit ] ^ a b c d Emmett T.APOE, UROS, WIPF1, XPA, XPC, SMC1A, TP63, SMC3, TBX4, TRAF3IP2, GJB6, PLXND1, PIGN, NIPBL, TINF2, MBTPS2, RTEL1, WRAP53, SETD5, NOP10, SLC39A4, NHP2, HDAC8, USB1, CTC1, WAS, TERT, MMP1, ERCC2, ERCC4, ERCC5, GATA1, GNAQ, DKC1, KMT2A, TERC, DDB2, CST6, PARN, PKP1, PLCD1, RAD21, REV3L, MSMO1, COL7A1, ST14, STAT1, ADAM17, ERCC3, LTB4R, IL4, MMP9, PSIP1, CX3CL1, LACRT
-
Focal Segmental Glomerulosclerosis
Wikipedia
These include: NPHS1 , which encodes the protein nephrin that contributes to the filtration barrier; [25] NPHS2 , which encodes the protein podocin found in podocytes; [26] and INF2 , which encodes the actin-binding protein formin . [27] Diagnosis [ edit ] Diagnosis of FSGS is made by renal biopsy that includes at least 15 serial cuts with at least 8 glomeruli. [28] [29] Histologic features include sclerosis (scarring) of a portion (average: 15%) of the glomerular space, with only a portion of glomeruli manifesting any sclerosis. [30] Other tests helpful in the diagnosis include urine protein, urinalysis, serum albumin, and serum lipids. [2] A clinical picture of proteinuria, low blood protein levels (albumin, antibodies), and high blood cholesterol would support a diagnosis of FSGS, although these do not help to distinguish between FSGS and other causes of proteinuria . [5] [9] Classification [ edit ] Micrograph of the collapsing variant of FSGS (collapsing glomerulopathy).TRPC6, INF2, AGT, NPHS2, ACTN4, CD2AP, APOL1, SERPINE1, PAX2, CRB2, MYO1E, ANLN, MYH9, SGPL1, NPHS1, SYNPO, TGFB1, EDN1, ZMPSTE24, COL4A5, NXF5, LMX1B, ARHGAP24, CCN2, LMNA, MPV17, NOS2, PCNA, FN1, E2F3, ACTA2, GNAQ, HAVCR1, SPP1, MYH10, COL4A1, AGTR1, MMP9, MMP2, LIPC, AGTR2, CASP9, ANGPT2, VLDLR, LPL, ACTR3, PIAS1, WT1, REN, PLCE1, SMARCAL1, COQ6, NUP107, PTPRO, TBC1D8B, ITGA3, COQ8B, SCARB2, CLCNKB, NARS2, NUP160, NUP205, WDR73, SLC12A3, NUP133, COL4A4, NUP93, ACTB, SEC61A1, VPS33A, CLCN5, IGAN1, TRIM8, G6PC, SLC37A4, ACE, NUP85, COL4A3, CD44, VEGFA, TNF, TRPC5, CCL2, POMC, PODXL, TBPL1, TYRP1, ALB, RAC1, NFE2L2, BAX, CAMK4, CCR2, APOE, KRT20, MAPK1, MS4A1, PON1, PLG, CD80, TLR4, MAPK3, IL10, MTOR, MIR193A, CYP11B2, LAMB2, HLA-A, IL1A, IL1B, UBD, HPSE, C1QL1, NES, CHP1, GDF15, POLDIP2, MAFB, AHSA1, SIRT1, YAP1, RAPGEF5, BMS1, RNF19A, PLA2R1, MLYCD, DDN, KEAP1, PTPRU, MMRN1, SERPINA3, SMPDL3B, SIRPA, CNKSR3, RHOV, SDK1, THSD7A, NPNT, NPHP4, LINC01193, MIR106A, MIR150, MIR155, MIR19B1, MIR200C, MIR30A, MIR34C, HNP1, MIR451A, MIR490, MIR663A, KTWS, MIR1225, MIR1915, PLB1, HT, IGKV3-7, PLEKHH2, IL22, DERL2, ATL1, ZDHHC2, WT1-AS, SNX9, NBAS, ADA2, KIRREL1, FBXW7, PLXNA3, RPL23, PDSS2, TMEM63C, VANGL2, RBPJP4, PDLIM2, HHIP, TTC21B, ALG13, STRIP1, GRAP2, CCL19, KLF4, CFH, DMPK, ECT2, EGF, ENPEP, EPHB2, EYA1, F2RL1, GABPA, GH1, CXCL1, HLA-DQA1, MAGI1, HMOX1, HP, HTC2, IL1R1, IL6, IL18, ILK, ITGAE, ITGB1, LAMA5, CTLA4, VCAN, CSF2, MAPK14, ANK3, FASLG, B2M, BAK1, BCL2, BCL2L1, BSG, C3AR1, CALB2, CD9, CD40, CD40LG, CDC42, COL1A1, COL8A1, COL8A2, KLF6, COX10, CP, CRH, CRK, LAMC2, LCN2, LPA, ACACA, SLC5A2, SLC12A1, SNRPB, TAGLN, HNF1B, TGFBR2, TIMP1, TLR2, TYRO3, UBA52, KDM6A, VASP, YWHAZ, PAX8, CXCR4, MANF, AIMP2, CUBN, CASK, NRP2, PDLIM1, SELP, SCN7A, SMAD7, S100A4, MMP14, MTHFR, TRNL1, MUC1, MYH1, NAGLU, RPL10A, NEFH, NOS1, NOTCH1, NPHP1, PKD1, PKD2, PLA2G1B, PLAU, PLCG1, PPARG, PTPRC, RAP1GAP, RARA, RPL17, LOC105374325
-
Abortion In Virginia
Wikipedia
That year, 78% of women in the state aged 15 – 44 lived in a county without an abortion clinic. [24] In 2017, there were five Planned Parenthood clinics in a state with a population of 1,971,590 women aged 15 – 49 of which four offered abortion services. [25] Statistics [ edit ] In the period between 1972 and 1974, the state had an illegal abortion mortality rate per million women aged 15 – 44 of between 0.1 and 0.9. [26] In 1990, 745,000 women in the state faced the risk of an unintended pregnancy. [22] In 2010, the state had two federally funded abortions. [27] In 2013, among white women aged 15–19, there were abortions 1090, 1280 abortions for black women aged 15–19, 250 abortions for Hispanic women aged 15–19, and 190 abortions for women of all other races. [28] In 2014, 55% of adults said in a poll by the Pew Research Center that abortion should be legal in all or most cases. [29] In 2017, the state had an infant mortality rate of 5.9 deaths per 1,000 live births. [9] Number of reported abortions, abortion rate and percentage change in rate by geographic region and state in 1992, 1995 and 1996 [30] Census division and state Number Rate % change 1992–1996 1992 1995 1996 1992 1995 1996 South Atlantic 269,200 261,990 263,600 25.9 24.6 24.7 –5 Delaware 5,730 5,790 4,090 35.2 34.4 24.1 –32 District of Columbia 21,320 21,090 20,790 138.4 151.7 154.5 12 Florida 84,680 87,500 94,050 30 30 32 7 Georgia 39,680 36,940 37,320 24 21.2 21.1 –12 Maryland 31,260 30,520 31,310 26.4 25.6 26.3 0 North Carolina 36,180 34,600 33,550 22.4 21 20.2 –10 South Carolina 12,190 11,020 9,940 14.2 12.9 11.6 –19 Virginia 35,020 31,480 29,940 22.7 20 18.9 –16 West Virginia 3,140 3,050 2,610 7.7 7.6 6.6 –14 Number, rate, and ratio of reported abortions, by reporting area of residence and occurrence and by percentage of abortions obtained by out-of-state residents, US CDC estimates Location Residence Occurrence % obtained by out-of-state residents Year Ref No.
-
Lactic Acidosis
Wikipedia
Both L and D isomers of lactic acid are produced in the rumen; [21] these isomers are metabolized by different metabolic pathways, and activity of the principal enzyme involved in metabolism of the D isomer declines greatly with lower pH, tending to result in an increased ratio of D:L isomers as acidosis progresses. [25] Measures for preventing lactic acidosis in ruminants include avoidance of excessive amounts of grain in the diet, and gradual introduction of grain over a period of several days, to develop a rumen population capable of safely dealing with a relatively high grain intake. [21] [22] [23] Administration of lasalocid or monensin in feed can reduce risk of lactic acidosis in ruminants, [27] inhibiting most of the lactate-producing bacterial species without inhibiting the major lactate fermenters. [28] Also, using a higher feeding frequency to provide the daily grain ration can allow higher grain intake without reducing the pH of the rumen fluid. [29] Treatment of lactic acidosis in ruminants may involve intravenous administration of dilute sodium bicarbonate, oral administration of magnesium hydroxide, and/or repeated removal of rumen fluids and replacement with water (followed by reinoculation with rumen organisms, if necessary). [21] [22] [23] References [ edit ] ^ Woods, Hubert Frank; Cohen, Robert (1976).PDHA1, TRNL1, DNM1L, RRM2B, ND6, ATAD3A, PLAT, CYTB, ACAD9, MTO1, PDP1, POLG, BCS1L, PC, ISCU, COX1, ND1, ATP5F1E, SCO2, RMND1, TMEM70, COQ9, ND4, KARS1, FBXL4, TRMT10C, NDUFB11, LIPT1, MRPS14, NDUFS4, SLC25A3, EXOSC9, WARS2, SUCLA2, ELAC2, SURF1, TUFM, HADHA, IBA57, SLC37A4, DLD, EARS2, NDUFAF3, MICOS13, NDUFA11, NDUFAF6, NAXE, MLYCD, LARS2, FASTKD2, SFXN4, TANGO2, USP18, TPK1, ETHE1, FARS2, LIAS, NFU1, SLC25A42, NDUFS7, LIPT2, SLC25A13, LRPPRC, TSFM, COA6, BOLA3, PREPL, UQCC3, SUCLG1, PDHX, UQCRFS1, COQ2, MRPS34, COX20, SLC25A19, NDUFAF5, AARS2, LYRM4, COQ8A, TMEM126B, CAMKMT, AGK, TRMU, NUBPL, PUS1, FOXRED1, COA8, TK2, GTPBP3, SERAC1, COX14, LYRM7, ATPAF2, MPC1, NDUFAF2, LARS1, TIMMDC1, TACO1, NDUFAF1, MRPS7, YARS2, MRPS16, PNPLA8, TIMM50, NDUFAF4, PET100, TRNN, TRNS1, NDUFS2, NDUFS1, NDUFB10, NDUFB9, NDUFB3, NDUFA10, NDUFA6, GFER, NDUFA1, TRNW, TRNV, TRNT, TRNS2, TRNQ, NDUFS3, HSD17B10, HADHB, TRNK, TRNH, TRNF, ND5, ND3, HADH, ND2, COX3, COX2, TRNC, MPV17, G6PC, NDUFV1, CYC1, NDUFS6, SLC25A4, ALDOB, ATP5F1D, BCKDHA, SLC3A1, BCKDHB, CA5A, CHAT, COX6B1, SDHA, SCO1, COX8A, COX10, COX15, MIPEP, DBT, PPM1B, DGUOK, PHKG2, DLAT, PHKA2, PDHB, PCK1, PCCB, PCCA, TYMP, FH, NDUFV2, NDUFS8, HSPA5, MB, TFAM, TXNIP, VARS2, CA2, PTGS1, RPS6KA3, ABCB6, MGAM, SRSF2, SI, MEHMO, MAP2K7, SLC5A2, UQCRB, ALDH7A1, ATF4, AQP9, LINC01194, TTR, PTBP1, ARRDC4, MET, GSTZ1, IL10, IL6, MRPS22, SLC47A1, TNF, ZDHHC13, NDUFA5, RNPS1, UQCC2, UQCRQ, OPA1, FBP1, ECHS1, TRS-AGA2-3, ALB
-
Dysphagia
Wikipedia
Journal of Neurology, Neurosurgery, and Psychiatry . 75 Suppl 3: iii22–28. doi : 10.1136/jnnp.2004.045906 .MEGF10, TSEN2, TSEN54, TSEN34, KIT, ATXN3, MATR3, ATXN1, PANK2, TWNK, SLC52A2, PSEN1, TP63, DYSF, AR, DNAJB6, ATXN2, ATXN8OS, SCN4A, PABPN1, CACNA1A, SLC9A6, KLHL41, TUBB4A, NOP56, SON, IKZF1, STUB1, HUWE1, DNM1L, SCO2, KMT2B, SEMA3E, SNCAIP, OPTN, ERLIN2, NPC2, AFG3L2, PIK3R5, KAT6B, MACF1, TARDBP, ATP13A2, SPECC1L, DNAJC13, AGTPBP1, DDHD2, VPS13A, PLXND1, SPART, SETX, DKK1, PUF60, POLG2, POLR3A, MYOT, CACNA1G, MED17, SURF1, TPM3, TPM2, TOP3A, TK2, TBP, TBCD, VAMP1, SPG7, VARS1, SOX4, ACOX1, SNCA, SNAP25, SLC25A1, SLC18A3, SLC6A9, UBTF, VCP, CYP7B1, SYNJ1, PEX16, NRXN1, PLAA, TRIP4, PIGQ, NUP62, SQSTM1, PDE8B, WFS1, PRKRA, KCNAB2, CNTNAP1, PLA2G6, COLQ, SMC1A, XRCC1, PIGN, CHMP2B, FBXO7, PYROXD1, REPS1, LINGO1, TUBB6, C19orf12, SLC19A3, FLAD1, SPG11, KIAA0319L, SLC25A22, MGME1, RHBDF2, CARS2, CDC73, FA2H, REEP1, IRF2BPL, PRDM16, SLC5A7, NDUFAF2, SLC52A3, ATXN10, RNASEH1, CHCHD10, MYMK, AGRN, KY, CAVIN1, KANSL1, SYT14, EBF3, C9orf72, LRRK2, ARX, KCTD7, SIK1, CARMIL2, TTBK2, HGSNAT, KLHL40, SYT2, GBA2, ALS2, ARID1B, TBK1, YARS2, ASCC1, GEMIN4, RRM2B, UBQLN2, LRP12, GMPPA, ANKRD11, SACS, MYORG, FBXL4, TOR1AIP1, GIGYF2, CNTNAP2, SETBP1, SDHD, PNKD, NDUFAF3, MECR, TRAPPC12, COQ4, RLIM, SELENON, MFF, LMOD3, KLHL7, VPS11, TBC1D23, VPS35, POLR3B, VAC14, CHD7, FERMT1, NCAPG2, GDAP2, PDP1, MAGEL2, SPG21, KCNK9, SKI, ATXN8, KCNC3, KCND3, ITGB4, CACNA1C, IRF6, IRF5, CAV1, IDH2, IDH1, HPRT1, HOXB1, HPCA, HLA-DRB1, HLA-B, HTT, H3-3A, GUCY1A1, SDHC, KIF5A, CHAT, LAMA2, ATP6, MRE11, MPZ, MMP1, KMT2A, MID1, MECP2, MCL1, MAPT, BRAF, LMNB1, LIFR, LBR, SERPING1, LAMB2, SCARB2, GRM1, ND2, EPRS1, CHRNE, ECM1, TYMP, ATN1, CLCN1, CCR6, DYNC1H1, DMPK, COL7A1, DGUOK, TIMM8A, COL13A1, CYP27A1, CTNS, CCN2, EIF4G1, FMR1, GRIN2D, FXN, GRIN2B, GNS, GNAQ, GNAO1, GLUD2, GJB1, GFPT1, GCH1, CHN1, CHRNA1, GBA, GABRD, CHRND, FUS, FTL, ND1, MSX1, ND3, OPA1, ADH1C, PRNP, MAP2K2, PRKCG, POLG, SLC25A4, EXOSC9, PLP1, PLEC, PIGA, PFN1, APP, PDGFRA, ND4, ASPA, PRPS1, PSAP, ADD3, ATXN7, SDHB, ACTA1, SDHA, SCN8A, ACTB, SCN3A, RYR1, PTCH1, REV3L, ACTG2, ADAR, QDPR, NECTIN1, PTS, RERE, PCNA, NR4A2, NDUFA9, ATP7B, ND5, ND6, TRNK, TRNL1, TRNV, TRNW, ATP7A, MYH7, MYH8, MYO9A, NDUFA6, ATP1A3, NEB, CRYAB, NDUFS3, NDUFS1, NPC1, NF1, NEUROD2, NEK1, NDUFS2, NDUFB8, SOD1, BDNF, TAC1, CRP, ALB, MORC3, NPNT, ATP4A, ATP12A, COPD, IGFALS, BMP2, ARSA, CRYGD, ASXL1, UCN2, MALAT1, APOE, CA3, IS1, OTULIN, SGCA, CSF2, NBN, NT5E, PAFAH1B1, CDK13, PAX9, ELP1, SRPX, EPX, ZP3, YWHAE, PNN, TRI-AAT9-1, TRNAG1, TPO, PSMA7, RAD51, TRIM21, RPS20, SLN, SLC10A1, PCYT1B, SAE1, CTBS, CCL26, ECI1, DMD, DPP4, MRS2, EGF, FUT3, GCG, TET2, GSS, GSTP1, IL5, PART1, KRAS, TNFRSF13B, SF3B1, LAMC2, LY6E, MMP8, KDELR2, ACR
-
Neurofibromatosis
Wikipedia
However, radiotherapy is not recommended in children who present with this disorder. [28] It is recommended that children diagnosed with NF1 at an early age have an examination each year, which allows any potential growths or changes related to the disorder to be monitored. [29] Prognosis [ edit ] In most cases, symptoms of NF1 are mild, and individuals live normal and productive lives.
